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@hungryfatman2
Stuff me full of carbs and fats. I should be too full to move, at all times.
Imagine that you’re in your bra and panties. I come up from behind you, and press by belly into your back. You can feel the swiftness of my belly envelop you. I reach into your panties, and slowly trace my my index and middle finger, along the outside of your lips (no, not the lips, on your face).
As my hands move, I gently, dip in, and brush your clit, with the tip of my middle finger, and I whisper, into your ear, “I’m hungry.“
If you are concerned about how the recent U. S. federal court ruling, declaring the Affordable Care Act (Obamacare) unconstitutional, might effect your ability to get access to healthcare, I suggest checking out the Book, The Self Pay Patient.
Nearly everything I used to develop a healthcare plan B, when I was out of work, for an extended period of time, can be found in that book.
Outgrowing pants.
Yesterday, I was getting dressed for work, and found that I was barely able to button the size 50 w pants, I was planning to wear. With a bit of struggle, I was able to get them buttoned; but just barely. My size 52 w pants are getting a bit snug too. I recently got some size 54 w pants, to grow into, but I’m starting to think I should’ve gone with a bigger size.
Stuffed
I had been out of work for an extended period of time; far too long. I recently got back to work, and decided to get myself a little something extra with my first check. After work, I had 2 Sonic, breakfast toaster meals; one with Coke, and the other with a chocolate shake. It’s nice to be full like this. I wish I could do it every day.
#creations
A Feedee’s Thoughts On Staying Healthy, While Gaining - Part I
Part I Part II Part III Part IV Part V
I am a feedee. My earliest memories of having the desire to get fatter, was when I was about four years old. It wasn’t sexual for me then, but it became sexual for me, at about the time I hit puberty. Surprisingly, I wasn’t a fat kid. As much as I had the desire to get fatter, I also had the sense that I should resist it; and I did (for the most part); at least until after I became an adult.
The desire to get fatter intensified after I hit puberty, and resisting it intensified the desire even further. When I resist it, the desire builds up to a point I can no longer resist. I eventually wind up taking that desire out on whatever food I can afford to put away, and put on a lot of weight in the process.
When I don’t resist it, the weight comes on more slowly, and more healthfully. I allow myself to indulge, from time to time, and am able to enjoy the process more fully. Either way, the desire never fully goes away. I’ve come to see it as a sort of enjoyable curse. but after what it has taken to accept that part of myself, I wouldn’t be rid of it now; even if could.
I was also born with a bad heart valve, and it has had to be replaced twice. Once when I was twenty, and once just a few years ago. Both times I almost died; repeatedly. Both times, I got to hear myself flat line; which is a surreal experience, that I’d rather not repeat any time soon. (The upside is, I might technically be undead.) The second time it was replaced, was because the first, mechanical valve had an infection attached to it; that appeared to be building a brave, new, bacterial civilization. It was probably bad enough, by the time I knew something was wrong, that the valve would have had to have been replaced; even if it had been diagnosed correctly; early on.
Regardless of who screwed which diagnosis up, and prescribed drugs that made things significantly worse, and (I believe) wound up causing heart failure, I’m still around five years later, to write about it today. However, even after all that, my cardiologist recently told me that, even with the damage that has been done to my heart, I’d probably live for a very long time. I don’t know how long that is, but it’s already been longer than I expected. (...and as of my last testing, my ejection fraction 49% which is almost normal.)
I talk about death a bit in this post series. It’s important to recognize that it’s going to happen, whether we like it or not. We might be able to postpone it for a while, but death comes for everyone eventually. Maybe some of what I have to say will help some feedees kick death in the stones, spit in his eye, and send him on his way.
Living longer might be important, but it’s also important to live while you’re alive. Hopefully, some of what I’ve written in this series of posts will help a few feedees and non-feedist fatties to do both.
Why Would I Open Myself Up To Criticism By Mentioning All This?
Considering that publishing this is almost certainly going to cause the concern trolls to stumble out from under their bridges, so they can tell me how all about how being fat is bad for people, you might wonder why I mention any of this.
Eating trolls is great fun for me (I am bigger than you. Get in my belly.), and there are few things more fulfilling that giving a bully a swift kick to the teeth; or a good stomp on the head.
That said, I usually have better things to do with my time than mind wrestling with losers who need put others down, in order to feel good about themselves; and morons who genuinely think they are doing fat people a favor, by telling them all about how being fat is bad for them.
Truth is, I genuinely believe that I have something of value to say on the subject, that can not only improve the health and extend the lives of feedees. I also believe do the same for non-feedist fatties; who are done punishing themselves with weight loss strategies that don’t work for them. If that means I’ll have to eat a few concern trolls, as a result of posting this, I’m sure they’ll be delicious.
In order to establish a bit of credibility, so that the people who might benefit from what I have to say, might see the value in it, I believed I need to establish a few things.
I am a feedee, and being a feedee, for me, is not just a quirk or just a kink. If it isn’t hardwired into my DNA, it may as well be.
I am not in a hurry to die.
I have had reason to be highly motivated to find ways to reduce the negative impact on my health.
What I have learned has (or at least appears to have) had a positive effect on my health.
I’m fat, but not supersized, and while the health challenges I’ve had weren’t caused by me being a feedee, they can certainly be made worse by it.
I’m in no hurry to die, but I still want to live while I’m alive. My luck won’t last forever, but I have beaten the odds, when it comes to surviving health challenges that should have killed me; several times over.
None of those health challenges were caused by being fat, or by getting fatter; but I learned a lot of things about the possibility of being healthy while being fat and getting fatter; as a result of those challenges. Just as importantly, I’ve used what I’ve learned to improve and maintain my own health.
...and unless there’s something that can get my risk of dying below 100%, odds are, it’ll happen eventually. I’m in no hurry for it to happen though, and much of what I’ve learned has helped me to (mostly) recover from health issues that would have killed most non-feedists.
I’d hope that’s enough to convince you that my thoughts on the subject are, at least, worth considering.
The Secret Is In The Sumo
Not long ago, I re-read the article, “Why Don’t Sumo Wrestlers Die of Heart Disease?”
I read the article a while ago. It was interesting, but at the time, it didn’t seem as if it contained actionable information, that I didn’t already know.
A tl;dr version of the article could say something like, “Sumo wrestlers are huge, and I didn’t understand how they could be so fat, and still be healthy; until I read about a study that showed that people who exercise tend do be healthier than those who don’t; regardless of how fat they are.”
Here’s a particularly interesting snippet from the article:
“I inquired about their health status after retirement, and, to my surprise, they were pronounced hale and fit into their 70s and 80s. I did not have an explanation.
Received wisdom dictated that these people are sitting ducks for diabetes and heart disease.
The answer, finally!
have to admit that every time I preached the benefits of weight control and the penalty sinners would inevitably have to pay, I had this nagging doubt, What about those sumo wrestlers?
In an article published in JAMA, scientists from the University of South Carolina in Columbia looked into the issue of adiposity (fatness) vs. cardiorespiratory fitness as determinants of death.
In a 12-year study, researchers found that among 2603 American adults over 60 years old, those who engaged in cardiovascular activity were living longer than those who exercised less, even when they had the same amount of body fat.“
I didn’t have all the pieces to the puzzle, when I first read the article. There’s a good chance, I still don’t have all the pieces, but I do have more.
After re-reading the article, I decided to review, what I thought I already knew about the Sumo. (Why I was curious about them, in the past, should be obvious, to anyone who knows what a feedee is.)
This time, there were a few things that jumped out at me about the Sumo lifestyle, that jumped out at me as likely causes of their better than expected health, with their super sized bodies.
I’ll go into each of these in more detail, in part II. For now, though, I’d just like to point out a few of the things that jumped out at me; with a brief explanation about why they did.
They exercise. This one should be obvious.
They don’t eat until lunch time, or later. Some research shows that fasting for part of the day can be good for your health.
They exercise in a fasted state. Some research shows that exercising in a fasted state increases your insulin sensitivity.
They start feasting shortly after their morning workout. They feast while their fat cells are likely to be at peak insulin sensitivity.
Their post workout feast (Note that I said “feast”, and not “meal”) is most likely to be high in carbohydrates, low in fructose, and have plenty of vegetables. Large amounts of fructose is believed to be a key contributor to “weight related” health issues. I also that consuming large amounts of carbs, when your fat is at peak insulin sensitivity, makes it easier for your fat cells to clear your blood stream a lot of the stuff that modern medical science causes “weight related” health issues.
They sleep after feasting. I’m not so sure about this one, but there is some research that indicates this might help you grow new fat cells.
In parts II and III, I’ll go into what I suspect to be the core cause the four main things that I suspect to be the key drivers behind, what modern medicine calls “weight related” diseases. We have some degree of control over three of them, but there’s one that I’m not so sure that we can do anything about. But before I cover them, I’ll explain why you want to read all the way through to the end, rather than skipping to the last post, where I’ve posted a few ideas on how I think you can be healthier and live longer; while living the feedee life. I’ll also go into further detail, about why these aspects of the sumo lifestyle jumped out at me, and why growing new fat cells might be a good thing; even for those who’d prefer not to be fat.
Part II
A Feedee’s Thoughts On Staying Healthy, While Gaining - Part II
Part I Part II Part III Part IV Part V
I wish I could say that I have the secret to getting as fat as you want, while also living a long and healthy life, but I don’t. What I can say, however, is that I’ve learned a few things that I think can help feedees live a longer, healthier life.
Why is this important, besides the obvious reason? Most people have a fear of dying. Dying is the easy part. When it’s done, you have no more pain, and no more problems.
The fear, pain, suffering, and health care costs that happen while you’re still alive aren’t as easy to deal with. It’s also tragic for the people who have been left behind, and for the people who have had to watch you suffer.
On a more practical note, the fear, the pain, and the suffering, of dealing with a major health issue are no fun at all. If what I’ve learned can help extend a few feedee and non-feedist fatty lives, or reduce or postpone some of that suffering, I’d consider that a win.
Major health issues are expensive too. Healthcare is a finite resource, and whether healthcare is provided via market means or via political means, access to healthcare will be rationed in some way, shape or form. Either way, feedees are kind of fucked,
It’s no stretch of the imagination to think that some feedees will need access to more healthcare resources than non-feedist. Even if free markets were allowed to drive prices down, a feedee’s healthcare needs might still be too expensive to be affordable.
Even in a government run system, resources aren’t finite, and when resources are stretched thin, politically unpopular people are the first to be thrown under the bus. Like it or not, being fat is politically unpopular, and like smokers, fatties will likely be among the first to be forced to go without; when the politically driven rationing of healthcare starts.
If what I’ve learned can help save a few feedees and non-feedist fatties from suffering that fate, or help put it off for a while, or even help then save a little on their healthcare costs, I’d consider that a win too.
Why You’ll Want To Read All The Way Through To The End
I’ve put most of my recommendations at the end of this five part series at the end, but that’s kind of the dessert of the series. (Five parts, really? How the hell did it wind up being that long?!?)
The dessert is the best part of a meal. but the meal is part of what makes the dessert so good. It’s the same with the series. Parts II, III, and IV are kind of like the meat and potatoes (and vegetables, yuck!) of the series.
What I have to say at the end might make sense on it’s own, but it’ll make more sense, after you’ve read those parts. When you read the whole thing, you’ll not only know what you should do, but also why you should do it.
Now on to the meat and potatoes...
In part I, I mentioned that there were four key things that I suspect to be the driving force behind weight related disease.
Most of of those five things are, in some way, related to what I believe to be, the real core cause of what modern medical science calls “obesity related diseases”.
Over time, I have come to believe...
There Is A Limit To How Fast The Human Body Can Store Energy
In essence, we start running into problems, when we consume calories at a rate that is faster than our bodies can store them, and do so over an extended period of time. Then, instead of being burned as energy or stored as fat, the excess sugars, fats, and proteins that have been consumed continue circulating in the bloodstream; causing all sorts of shenanigans. There are reasons I believe this to be the case, and I’ll get to them soon. (I like the word, “shenanigans,” but I don’t know why. This gave me an excuse to use it.)
Granted, this is similar to the common belief that we, “eat too much, and we move too little.” However, there is a key distinction.
Conventional wisdom says that being fat is the problem. I am saying that the rate of energy storage, and storage capacity is the problem.
Conventional wisdom would say that means we need to move more, and eat less.
As a feedee, I say we need to find ways help the body store more of what we consume as fat, and to do it faster.
Considering that traditional medicine hasn’t done such a great job of helping people prevent (what they call) obesity related diseases, I’d say my way makes more sense; even for non-feedists; who’d prefer not to gain weight.
In part III, I’ll cover what I believe to be the four key drivers behind, what are considered to be “obesity related diseases.,” and no, obesity isn’t on the list.
Part III
A Feedee’s Thoughts On Staying Healthy, While Gaining - Part III
Part I Part II Part III Part IV Part V
In part II, I mentioned that I suspect that much of what is usually called obesity related disease, was caused not by obesity itself, but rather by limitations on how much energy we can store, and how fast we can store it.
In this post, I’ll continue on that path, and in the process, explore what I believe to be the four key drivers of obesity related disease.
These drivers are the number of fat cells you have and ability to grow new ones, insulin resistance, chronic inflammation, and the need for hormetic stimulus. (There has to be a better way to say, “the need for hormetic stimulus.”)
In this post, I lay the groundwork for part IV, where I’ll explore some of the things feedees and non-feedee fatties can do to maintain and improve our health. This is going to be a long post, but I’ll try to make it worth the read.
The Four Key Drivers of Obesity Related Disease
1. The number of fat cells you have. Beyond the fact that there are no diseases fat people get, that skinny people don’t, there are a couple of things that lead me to believe this is a key component of obesity related diseases.
I suspect that if you don’t have enough fat cells to store what you’ve consumed at a fast enough rate, that you’ll start running into the limit of how fast your body can store energy.
There are some people who have a disorder that causes them to have very little fat tissue, but all of the metabolic markers of what are considered to be insulin related diseases. [ link ]
Here’s a short snippet from the article that lines up with my suspicions:
They engineered mice that could make an almost limitless amount of fat tissue. As a result, there was no end to the amount of fat the animals could store. They were, Dr. Scherer said, “the fattest mice under the sun, the mouse equivalent of an 800-pound human being.”
The fat mice were metabolically normal. [emphasis mine]
Now, with years of research, the picture has become clear. And so has a new view of the role of fat itself in causing the medical problems of obesity.
At the heart of all these conditions and what is known as “metabolic syndrome,” or having at least three of the conditions associated with obesity, is an inadequate ability to store fat.
The experiment with mice that could make an almost limitless amount of fat tissue is particularly interesting. The mice that were genetically engineered to be better at storing fat, didn’t run into the metabolic problems that are considered to be “obesity related diseases.“
Of course genetically engineered mice aren’t humans, and even if humans can be genetically engineered to store almost limitless amounts of fat, that doesn’t help people who are alive today.
It used to be thought that adult humans could not grow new fat cells, but that believe has been found to be incorrect. [ actual study ]
I see the ability to deliberately grow new fat cells as a sort of feedee health, holy grail. I also believe it’d be beneficial to people who’d rather not be fat; even if they don’t like the side effect; of adding padding.
As far as I am aware, modern medical science has not found a way to grow new fat cells; on command. There are some clues to be found though, like this research that I believe shows (surprise, surprise, surprise) that being physically lazy causes fat cells to multiply. (my interpretation) [ link ]
Here a snippet of what I believe to be the relevant part of the article:
According to Prof. Gefen, a main question for the researchers — working through a grant from the Israel Science Foundation – was to find out why a sedentary lifestyle often results in obesity. “We found that fat cells exposed to sustained, chronic pressure — such as what happens to the buttocks when you’re sitting down — experienced accelerated growth of lipid droplets, which are molecules that carry fats.”
Apparently, once a cell gets “fat” by accumulating droplets of lipids, the structure of a cell and its mechanics change dramatically. Using an advanced atomic force microscope, the researchers were able to see how cells got stiffer as they expanded.
“When they gain mass and change their composition, expanding cells deform neighboring cells, forcing them to differentiate and expand,” said Gefen. “This proves that you’re not just what you eat. You’re also what you feel — and what you’re feeling is the pressure of increased weight and the sustained loading in the tissues of the buttocks of the couch potato.
I didn’t find the study that was referenced by the linked article, but what I gather from what is saying, they found that the pressure put on fat cells; from sitting or laying around, causes them to multiply.
So far, we know that the number of fat cells you have can increase during rapid weight gain [ link ], and that being physically lazy both appear to cause fat cells to multiply. If that was enough, though, there would be no such thing as obesity related diseases.
As far as I’m aware, there is no nutrient, herb, or pharmaceutical drug that causes fat cells to multiply, but if something like that exists, I’d like to learn more about it.
2. Insulin resistance. [ link ] It’s fairly well known that insulin is the hormone that enables the glucose from the carbohydrates that you eat to get into your body’s cells.
Since I suspect that most people in the feedist community have encountered a gorillian explanations of how insulin works, I’ll try to keep that part of the discussion short. ( One gorillian is my new favorite number.)
Insulin as a few different jobs, but I think the ones that are the most relevant to our discussion here, is that it tells fat cells to stop releasing fat into the blood stream, reverse course, and start pulling fuel (both fat and glucose) from the bloodstream, and saving it for later.
This is why insulin is considered to be the weight gain hormone, and why (at least partially) insulin injections tend to cause people with type 2 diabetes to gain weight.
I’ve seen some people, online, mention that when some diabetic feedees (type 2, I assume), and got on insulin injections, they were able to blow up like balloons.
If all that was needed to blow up like a balloon, and lead a reasonable healthy life, was to eat a lot, and take insulin injections, I’d say, “Why wait for diabetes, let me get started with insulin shots right now.”
If only it was that simple.
The short explanation of type 2 diabetes, is that you consume too much carbohydrate (no such thing as too much), that carbohydrate is turned into glucose (sugar) as it enters your bloodstream, and your pancreas releases insulin to drive the glucose into your cells.
In order to function properly, your body needs the amount of glucose that circulates through your bloodstream to stay within a certain range, and when it gets too high, your pancreas will produce extra insulin to bring it back down.
When there’s too much insulin in your bloodstream for too long, your cells start getting used to it, and stop noticing that it’s there. It’s kind of like how you get used to the scent of a room; after being in it for a few minutes. The smell is still there, but you’d either need to intensify it, or leave the room for a while, to be able to smell it again.
The gist of what happens is, your cells stop being able to “smell” insulin, and your pancreas has to produce even more insulin; before your cells will respond to it, and start using or storing the glucose that is in your bloodstream. Over time, the pancreas becomes overworked, and starts becoming less able to produce enough insulin.
That’s about the time that doctors need to intervene with diabetes medications, and eventually insulin injections.
The added insulin is great news for those of us who enjoy the process of fattening up. It tells the fat cells to pull the fat and sugar from the bloodstream; and to hold on to the fat they have already stored. The trouble is, it doesn’t fix the problem of the cells not being able to “smell” the insulin, and that problem tends to get worse over time.
Since this post isn’t intended to become Scary Feedist Stories To Tell in the Dark, I’m not gong to go over all the gory details of the not so fun things that insulin resistance and type II diabetes are linked to, but there are a lot of them. One might say, insulin resistance is linked to a gorillian other health issues.
(In the book never written, Scary Feedist Stories To Tell In the Dark a group of feedees gather ‘round a mountain of cupcakes, and chant “dia-beetus, dia-beetus,” while filling their bellies; in an attempt to summon Wilford Brimley).
The reason insulin sensitivity is so important to this discussion, though, is that we can have a degree of influence over the insulin sensitivity of our cells.
Ideally, as a feedee, I’d be able to selectively increase the insulin sensitivity of my my fat cells, so that my fat can gobble up all those precious calories, circulating through my blood stream; before my muscles have a chance to burn them off. I’m not aware of of anything that does this, but if there is, I’d like to know about it.
The typical recommendation for increasing insulin sensitivity, is to lose weight. Since weight loss is kind of the opposite of what most feedees are aiming for, I’ll explore some of our other options in part IV.
3. Chronic inflammation is believed to contribute to many of the leading causes of death. [ link ]
From the linked article:
Diseases Associated with Chronic Inflammation
Cardiovascular diseases (CVD). Inflammation is an integral part of atherosclerosis (recall that oxidized low-density lipoprotein cholesterol stimulates the inflammatory response). Circulating inflammatory cytokines are predictive of peripheral arterial disease, heart failure, atrial fibrillation, stroke, and coronary heart disease (Singh et al. 2011, Emerging Risk Factors Collaboration et al. 2010).
Cancer. Several studies have established links between chronic low-level inflammation and many types of cancer, including lymphoma, prostate, ovarian, pancreatic, colorectal and lung (Aggarwal et al. 2006).(Kundu et al. 2008) There are several mechanisms by which inflammation may contribute to carcinogenesis, including alterations in gene expression, DNA mutation, epigenetic alterations, promotion of tumor vascularization, and the expression of pro-inflammatory cytokines that have roles in cancer cell proliferation (Kundu et al. 2008, Balkwill 2009)
Diabetes. The infiltration of macrophages into fat tissue and their subsequent release of pro-inflammatory cytokines into circulation occur at a greater rate in type II diabetics than in non-diabetics (Pickup et al. 2000, Nappo et al. 2002, Ortega Martinez de Victoria et al. 2009). Pro-inflammatory cytokines clearly decrease insulin sensitivity (Bastard et al. 2006).
Age-related macular degeneration (AMD). An evaluation of 11 population-based studies encompassing over 41,000 patients demonstrated a clear association between elevated serum CRP levels (> 3 mg/L) and the incidence of late onset AMD (Hong et al. 2011). The risk of AMD in these high-CRP patients was increased over 2-fold compared with patients with CRP levels < 1 mg /L.
Chronic kidney disease (CKD). The chronic, low-grade inflammation in CKD can lead to the retention of several pro-inflammatory molecules in the blood (including cytokines, AGEs, and homocysteine) (Glorieux et al. 2009). The reduced excretion of pro-inflammatory factors by the diseased kidney can accelerate the progression of chronic inflammatory disturbances elsewhere in the body, such as the cardiovascular system.
Osteoporosis. Inflammatory cytokines (TNF-α, IL-1β, IL-6) are involved in normal bone metabolism. Osteoclasts, the cells that break down (resorb) bone tissue, are a type of macrophage and can be stimulated by pro-inflammatory factors. Systemic elevations in pro-inflammatory cytokines push bone metabolism towards resorption, and have been observed to induce bone loss in persons with periodontal disease, pancreatitis, inflammatory bowel disease, and rheumatoid arthritis (Cao 2011). An increase in the levels of inflammatory cytokines is also a mechanism by which menopause stimulates bone loss.
Depression. There is a small, but significant association between elevated IL-6 and CRP in depressed patients, which has been observed in many population studies (Dantzer 2012). It is unclear whether inflammation leads to stress or vice versa, and there is data supporting both hypotheses (Gimeno et al. 2009) (Copeland et al. 2012).
Cognitive decline. Several observational studies have linked chronic low-level inflammation in older adults to cognitive decline and dementia, including vascular dementia and Alzheimer’s disease (Singh et al. 2011). One study found that people with the highest CRP and IL-6 levels (> 2.4 pg/mL) had a ~30-40% increased risk of cognitive decline compared to those with the lowest levels (< 1.4 pg/mL). (Yaffe et al. 2003). Inflammatory markers can be elevated before the onset of cognitive dysfunction, indicating their potential relevance as a prognostic tool in high-risk individuals (Singh et al. 2011).
Others. Elevations in circulating inflammatory cytokines are associated with several other conditions, both inflammatory (rheumatoid arthritis, IBD/Crohn’s disease, pancreatitis) and non-inflammatory (anemia, fibromyalgia, frailty, sacropenia/cachexia/muscle wasting) (Kaser et al. 2011) (Jha et al. 2009) (Ferrucci et al. 2010, Kadetoff et al. 2011, Rolland et al. 2011). Again, whether inflammation incites these conditions or results from them is unclear, and requires further investigation.
Page 1 of that same article provides us with a short list of causes of chronic inflammation, that can be summed up to say that chronic inflammation is caused by the foods you eat, and your metabolism of those foods. The foods you eat aren’t the only cause of chronic inflammation, but many food types that could considered to be feedee favorite foods, are foods known to cause inflammation.
4. The need for hormetic stimulus can loosely be summed up by the phrase, “use it or lose it.”
It’s a bit more complicated than that, but not by much. We’re built to resist environmental stressors. Even simply standing up, is an act of resisting an environmental stressor because, in doing so, you are moving in opposition to the force of gravity.
Hormesis [ more ] is mostly just a fancy way of saying that when you expose yourself to something, it can make you stronger.
The opposite is also true. You can see this in the way astronauts lose muscle and bone mass, when they’re in space. You can see it in the way the muscle on limbs shrink; when they’ve been placed in a cast; to heal a broken bone.
You can also see it in the effects of prolonged bed rest:
Effects on the heart and blood Like the muscular system, the cardiovascular system functions best when the body is in an upright position, working against gravity. After just a few days of bed rest, blood starts to pool in the legs. On standing, this can lead to dizziness and falls. Immobility also causes the heart to beat more quickly, and the volume of blood pumped is lower. The volume of blood generally in the body is lower, and there is less oxygen uptake by the body. This results in poorer aerobic fitness and fatigue sets in more easily. The blood also becomes thicker and stickier, which increases the risk of a blood clot forming, especially in the legs (deep vein thrombosis) and the lungs (pulmonary embolism). Effects on the lungs and blood Bed rest increases the risk of pneumonia and atelectasis (collapse of lung tissue). Fluid tends to build up in the lungs because the muscles aren’t working to remove excess fluid from the body. It’s harder for the lungs to expand when you’re lying flat, so blood pools in the chest area, leading to decreased lung volume. Coughing is not as effective due to weakened abdominal and chest muscles, causing mucus to collect in the lungs. Breathing also becomes shallower, which leads to poorer oxygen-carbon dioxide exchange in the lungs.
...
Effects on the metabolism and hormonal system Prolonged bed rest can cause numerous complex changes in the balance of hormones and minerals in the body, and in how the body processes energy. For example, immobility causes a reduction in the percentage of lean mass to body fat, and raises the risk of developing diabetes: immobile muscles can develop reduced insulin sensitivity, which in turn leads to raised blood sugar levels.
[ More on the effects of prolonged bed rest. ]
We’re built to move. We’re also built to sometimes have to go without food. However, most people in the developed world, rarely go more than a few hours without eating. This is where I think the sumo might be onto something.
In part IV I’ll continue along the same path, and discuss how feedees might be able to use exercise, occasional fasting, and diet to live the feedee life, and do so in a healthier way. The fasting and exercise tie in to the need for hormetic stimulus, but I won’t use that phrase going forward; mostly because I don’t like it.
Part IV
A Feedee’s Thoughts On Staying Healthy, While Gaining - Part IV
Part I Part II Part III Part IV Part V
I am not a doctor. As a feedee, I’d rather gain fat, than lose it. However, I’d also rather be healthy than sick, and I’m in no hurry to die.
This has lead me to do a fair bit of research on how I might be able to reduce the risks of being fat, and getting fatter. As far as I am aware, none of what I am about to suggest of people deliberately staying fat and getting fatter; over an extended period of time.
They are my best guesses at things that might help reduce the risks of living as a feedee. Given enough time, everyone’s risk of death is 100%, and nothing I say here will change that. The whole purpose of this, is to be able to live as a feedee, while also (hopefully) having a longer life; with less of the suffering and healthcare costs are currently associated with obesity.
That said, it’s still important to live while you’re alive.
Revisiting the Sumo
In part I, I said, the secret is in the sumo. It’s easy to see why a feedee would be interested in the sumo; even if he wasn’t health conscious. They’re one of the few groups of people who deliberately fatten themselves up to supersized weights, and are open about doing so.
More importantly though, they’re relatively healthy, compared to what modern medicine tells us to expect people weighing 400 lbs or more to be. I strongly suspect that the secret to their good health is in a few key lifestyle factors that help to keep insulin resistance and inflammation in check.
(Cholesterol and triglyceride levels appear to be related to blood sugar levels and insulin resistance in such a way, that I have come to see high cholesterol and triglyceride levels as usually being symptoms of insulin resistance. [link] [link] )
I’m no sumo expert, and I’m not a fan of the sport. The little bit (I think) I know comes from what I’ve read about them.
Based on what I do understand, though, here’s the gist of what a typical day for a sumo wrestler looks like: (For simplicity, I’ll just refer to this sumo as, “he.”)
He gets up early.
He does not eat breakfast.
He does a few chores.
He exercises. His training regiment would be grueling, for even seasoned athletes.
He has his first meal at, around lunch time; eats as much as he can; and drinks a lot of beer.
After his first meal, he takes a nap, and has some free time.
He has his second meal, eats as much as he can, and drinks a lot of beer.
He has more free time.
He goes to bed.
There are some things about this routine that seem like they’d be on a list of things that feedees shouldn’t do. The exercise is the obvious one, but you might also notice that the sumo is in a fasted state for about half the day. It’s less obvious, but you might also notice that he exercises in a fasted state.
One thing about the routine that stands out to me, is how strikingly similar it is to the way a person might use exercise and intermittent fasting to lose weight.
It’s odd that a routine that is effective for weight gain would be so similar to routines that are effective for fat loss. I suspect that insulin sensitivity is piece of the puzzle, that makes the whole thing work.
On Exercise
Yeah, I get it. A feedee’s knee-jerk reaction to the word, “exercise,” might be “yuck,” “eww,” or, “hell no!” Just hear me out, on this, for a minute. Nothing that I post here will make you skinny.
As a feedee, I’d much rather have a small army of feeders; catering to my every need; making sure I don’t have to lift a finger; so that every precious calorie that I can cram into my overstuffed belly works to make me just a little fatter. After all, I wouldn’t want to unnecessarily burn extra calories, by getting up to do things for myself.
Ok. I’m back.
Like it or not, exercise can work as medicine and helps to reduce health risks. [link]
Considering the focus I’ve placed on insulin sensitivity so far, it probably won’t surprise you when I say, exercise helps to increase insulin sensitivity. [link] [link]
The good news is, it’s possible to enjoy, at least, some of the health benefits of exercise, without losing weight. [ link ] And the sumo show that it is possible to gain weight, even if you get a lot of exercise.
I don’t know what the right amount of exercise would be. The CDC recommends people get 150 minutes of exercise per week; which works out to be five 30 minute workouts each week.
The feedee in me says no exercise is the perfect amount; zero minutes of exercise is exactly the right number of minutes of exercise.
Despite appearances, I don’t dislike exercise. (Weird, I know.) However, my aim with exercise is to get the greatest amount of health benefit I can, in exchange for the fewest number of calories.
In my searching, I came across the book, The First Twenty Minutes. The book provides a lot of interesting tidbits of information, but the core argument was that you receive the greatest benefit from the first 20 minutes of exercise. the author points out that there are benefits to continuing beyond the 20 minute mark, but with diminishing returns.
If you’re willing to put up with the weight loss talk, this article and audio interview are worth checking out. [ article ] [ interview ]
Here’s a couple of interesting quotes from the linked article:
The first 20 minutes of moving around, if someone has been really sedentary, provide most of the health benefits. You get prolonged life, reduced disease risk — all of those things come in in the first 20 minutes of being active.
Without being evangelical, I wanted people to understand that this is a book about how little exercise you can do in order to get lots and lots of health benefits.
You can always do more. But the science shows that if you just do anything, even stand in place 20 minutes, you will be healthier.
I usually get about 20 minutes of exercise in, 3 days each week, but it works out to be a bit more; because I stretch afterward, and do other physical activities that I don’t count as exercise.
If you’re just getting started, don’t start with 20 minutes. Start with 2 minutes, and then add a minute to each workout; until you build up to 20 minutes. If you’ve been inactive for a while, you’re body will need a bit of time to adapt to new physical activity.
Most articles and blog posts recommend talking to a doctor before starting an exercise routine. Since exercise stresses the body, that new stress can sometimes make an already existing problem worse; so it is a good idea to check with a doctor; before getting started.
If you’re especially heavy (supersized), and sometimes even if you are not, a lot of walking can cause unpleasantness, like plantar fasciitis; which makes standing and walking hurt like hell.
My usual exercise of choice is the stationary bike, but I’ll also do other things as well. Yoga, qi gong, and tai chi are all good options that are worth exploring.
If you’re heavy enough that it limits your mobility, there are sill things you can do. To start, there’s chair aerobics, chair tai chi, and chair qi gong. I’ll post a few links here, in case you need some more ideas. [link] [link] [link] [link] [link] [link]
Whether or not you’re interested in adopting a formal exercise program, you might want to consider making it a practice to simply stand up, or walk around for a couple of minutes; periodically, throughout the day.
Even if you just do that, it’ll provide you with some of the health benefits of exercise.
On Diet.
The short answer to the question, “What should a feedee eat,” is everything. You should eat the kinds of foods that motivate you eat a lot.
However, if you’ve taken a look into how the sumo fatten up, you might notice they don’t eat a lot of junk food.
The sumo staple food is a food called chanko nabi. It’s such a core part of their diet, that it wouldn’t be an exaggeration to call it sumo chow.
It doesn’t have a set-in-stone recipe, but it’s mostly made up of meat and vegetables. When they eat meals though, they don’t just eat chanko nabe, they also fill up with lots of rice, and (as I understand it) they drink plenty of beer; along with their meals.
What the sumo diet shows us, is that you can put on a lot of weight by eating large quantities of foods that are considered to be healthy.
The sumo are athletes. They’re fat, but the also carry a lot of muscle under that fat. With their grueling training regiment, they’re going to need a lot more protein in their diet than a typical feedee would.
The body uses protein to repair itself and build muscle, but excess protein can also be turned to fat. Gaining fat from protein is less efficient, because the body has to convert the protein to glucose, and then turn the glucose as fat; before it can be stored as fat. Too much protein in the diet can also place an unnecessary strain on the kidneys.
Carbs and fats are much more efficient at driving fat gain. An ideal fat gain diet would be a high carb, high fat, low protein diet. If you’re not looking to carry unnecessary, calorie burning muscle, a mostly low protein diet might be worth considering.
On a low protein diet, you’d be likely to be gaining fat and losing muscle at the same time; which means you could be getting fatter; even if your weight stays the same for a while. [link]
An interesting nugget of info from the linked article:
Throughout the course of the study, researchers also measured changes in the participants’ energy expenditure, or how many calories they used in a day, and found that those on normal- and high-protein diets were not only burning more calories than they did before the start of the study, but they were also using more significantly more energy than the low-protein group.
Meanwhile, resting metabolism in the low-protein eaters declined: in other words, as they gained weight, they required fewer calories to maintain that weight — a handy recipe for obesity.
In part, the difference can be attributed to the fact that the normal- and high-protein groups had gained more weight overall and needed more energy to move their bigger bodies around. Also, protein takes a lot more energy for the body to process — whether it’s being excreted or put toward building muscle or other lean body mass, which itself requires more energy to maintain — than fat. Fat, on the other hand, is much more easily stored as fat.
I’m not saying you should lay off the protein entirely. This isn’t going to stop me from eating ribs or buffalo wings. (I love buffalo wings.) It’s just something to keep in mind, and you choose what to eat.
I will, however, say that if you’d rather gain fat than muscle, it might be better to get most of your calories from carbs and fats than from protein; most of the time.
It’s probably also a good idea to be sure to include some fruits and vegetables in your diet. I don’t know how much is the right amount, but the standard recommendation is 5 servings of vegetables and 4 servings of fruit. [link]
I understand the desire to cut out the fruits and vegetables so that you have extra room in your stomach; that you can fill with high calorie goodness, but fruits and vegetables have important nutrients that you’ll probably miss out on; if you don’t make it a point to include them in your diet.
Maybe there’s a way to make a super-fattening, high calorie vegetable smoothie; that is digestible, and actually tastes good. If there is, I’d like to hear about it.
Fasting and Feasting
Combing fasting with feedism is a bit counterintuitive, but you can use fasting to help you gain weight. I know; I’ve done it. I’ll tell you more about what I did, and what the results were in a moment, but first, I’d like to talk a bit about fasting and health.
Using fasting as a tool to minimize the risk of weight gain is even more counter intuitive. I believe that you can. I’m not aware of any research that supports or refutes this idea, but there are some things we can be reasonably sure about.
When I first started to hear about intermittent fasting as a tool for heath, I thought it was just another silly fad. Even after I saw several doctors writing on the potential health benefits, I didn’t pay much attention to it. Part of that was because I thought it was just a fad, but it was also because it was being sold as a tool for weight loss.
It wasn’t until I caught wind of research that showed that people who fasted once a month had less heart disease. That’s what finally caught my attention, because it showed the potential for some real health benefit; even if it wasn’t used as the latest weight loss fad.
In addition to preventing heart disease, there’s also evidence that occasional fasting can help with the prevention and treatment of cancer. [link] [link] [link]
We’ve already established that sumo wrestlers tend to be healthier than we’d expect them to be; based on what modern medicine tells us about being fat. It’s also reasonably clear that their morning training exercises and the fact that they include plenty of vegetables in their diet play a major role in this.
It’s less obvious from the writings about their training, but we’ve also established that they’re in a fasted state for approximately half the day, or more.
I’m using the phrase, “intermittent fasting,” because it most accurately describes what I’m talking about. It’s usually used as a weight loss tool, but the the sumo lifestyle indicates that it can also be used as a weight gain tool.
There are a number of ways to do intermittent fasting, but most of them involve consuming no food, and only calorie free beverages for twelve hours, or more. For our purposes, this is what I mean when I refer to, “being in a fasted state.”
For most feedees, intermittent fasting can be as simple as skipping breakfast, like the sumo do, but I suspect that you need to be in a fasted state for longer, to really reap the rewards of fasting.
The concept of intermittent fasting includes time restricted eating, alternate day fasting, and the 5:2 diet. There might be other ways of doing it, but those are the ones that come to mind.
Time restricted eating involves restricting your eating to a 12, 8, or 4 hour time window; during the day. Alternate day fasting, involves a full (or nearly full) 24 fast, every other day. The 5:2 diet involves fasting 2 days out of each week.
There are a number of health benefits that nave been attributed to intermittent fasting, but I think the ones that are the most relevant to the needs of feedees are reduced inflammation, improved insulin sensitivity, reduced cancer risk, and improvements in heart health.[ link ] [ link ] [ link ] [ link ]
On top of all that, intermittent fasting has been shown to reduce inflammation.
I suspect that a big part of the reason that intermittent fasting works so well for both weight gain of sumo wrestlers and the weight loss of intermittent fasting enthusiasts has a lot to do with the way it improves insulin sensitivity. I’m not sure of it yet, but I also suspect that there is something about it that works to prepare fat cells to multiply, if you overeat; after the fast.
When I was first getting started with intermittent fasting, I started with one 24 hour fast each week. The reasons for the 24 hour fast, is that it was the one I was familiar with at the time, and it was the one that had already been shown to improve heart health.
My reason for choosing a once a week schedule was somewhat arbitrary. I had a vague recall of seeing a one day fast, once per week, having been recommended in the book, The Fasting Cure. It was a bit of a challenge at first, but got easier after a few weeks.
After I got used to them, the fasts did help me to feel healthier, in general. I had less nasal congestion, my airways were more open, I felt less pain, and my mind was sharper. As an added bonus, I was able to use the fasts to gain over 20 lbs, in a few weeks.
How I gained the weight was not unlike the way sumo wrestlers gain weight. When I broke the fast, I ate well past the point of fullness. On those days, I was able to eat far more than I would normally be able to.
Fasting and feasting in this way, had an interesting side benefit. My appetite on non-fast days was bigger. It started to take far more food on non-fasting days, just for me to feel like I wasn’t hungry, and it took even more food for me to feel somewhat satisfied.
I’m sure if I had pushed myself, even just a little, I could have gained a lot more weight, but economic constraints lead me to choose to hold back. I also have health challenges to deal with, and am concerned that such a rapid and deliberate weight gain would cause doctors not to work with me. It’d be nice to have feedee friendly doctors, but I think that might be too much to hope for.
As easy as it was to put on weight, I’ve also lost weight with intermittent fasting; without meaning to do so.
I suspect it has something to do with the way ketones and the increased insulin sensitivity effect the appetite. The gist is, when you are in a fasted state, your liver turns fat into ketones, because most brain cells can use ketones as an energy source; in place of glucose.
The ketones generated through fasting reduce your appetite; which makes it easy to feel more satisfied with less food. However, I suspect that there is also a mechanism in the body, that is triggered by deliberate overeating, after a fast, that tells your body that you need to eat as much as you can; and store it all as fat.
Maybe it’s a supercharged version of, what fitness and wight loss gurus like to call, “starvation mode.” I don’t know if that’s the case, but if it is, the name is fitting.
Either way, there seems to something about intermittent fasting that makes it a little too easy to lose weight (for a feedee), when you eat normal amounts of food; but also makes rapid weight gain easy, when you combine intermittent fasting with intermittent feasting.
In part V, I’ll wrap things up, and make a few health suggestions; based on my best guesses about how someone might minimize the risks that come with living the feedee life.
Part V
A Feedee’s Thoughts On Staying Healthy, While Gaining - Part V
Part I Part II Part III Part IV Part V
My main reason for putting this series together was to share some of what I’ve learned from the research I’ve done; in an attempt to to find ways to be healthier and live longer; while living as a feedee.
You might even say, I’ve been seeking ways that I might be healthy and live longer, while getting as fat as I want.
I don’t have all the answers, and I don’t know if such a thing is even possible. However, I do believe that feedees and fat people who aren’t feedists can have better health and longer lives; with a few tweaks to how they go about their business.
I hope others will find some of what I’ve written in these posts useful. Most information and research about health, diet, and weight focuses on people who want to lose weight. As far as I’m aware, there’s little or no information available that comes from the perspective of being fat and getting fatter in healthier ways.
I also hope that what I’ve pieced together can be used both by feedees who are happy to get fatter, and non-feedist fatties; who struggle with their weight.
I’m not a doctor, and I’m not a medical researcher by trade. I’m just a guy, on the internet, who has a knack for coming up with unique solutions to complex problems.
Hopefully, someone who is an actual medical professional can improve upon what I’ve pieced together, and come up with better ways for feedees to maintain our health; while being fat, and getting fatter.
The recommendations I’m about to make could be very wrong, but I do think I’m onto something; otherwise I wouldn’t have taken the time to write these posts. However, keep in mind that I am not a doctor, and even with as much as I’d like to think I know, there’s a chance that i don’t know what the hell I’m talking about.
Be sure to do your own homework on the subject, seek out proper medical advice, and use your own best judgement; before attempting any of what I suggest.
Two Steps Forward. One Step Back.
Bear with the fitness and weight loss talk for a minute. I have a point, and I’ll try to get to it quickly.
Shortly after high school, I got heavily into fitness training. The reasons and my past experiences as a gym rat are aren’t relevant to this discussion. What is relevant, however, is that I learned a thing or two about putting on muscle, and losing fat.
When a person wants to gain muscle they have to work their muscles, but they also have to let them rest.
When a person wants to lose fat, they have to consume fewer calories than they burn off, but if they stick to the low calorie diet all the time, their body adapts to it; and they stop losing weight. The way to counter this tendency, is to modify their diet in such a way that they are consuming fewer calories than they burn off; most of the time, but not all of the time.
I suspect that similar, possibly yet to be discovered, thing is necessary for healthier fat gain. I suspect that fat tissue needs to be given a bit of recovery time. The gist of the idea is to do nearly the opposite of what works for fat loss. Consume more calories than you burn off most of the time, but not all of the time.
That’s where intermittent fasting comes in. I’ve already discussed how fasting has been shown to improve insulin sensitivity.
In addition to improving insulin sensitivity, I also strongly suspect that fasting works to prime the weight gain pump, by getting ready to create new fat cells. For the moment, I don’t have a way to prove or disprove this hypothesis, but it’d explain why I gained weight so easily, when feasting after fasting.
It’d also help explain why sumo wrestlers tend to be healthier than modern medicine tells us they should be.
I suspect that it “primes the pump,” but that fat cells only multiply afterward; if you follow the fast up with a lot of over eating.
Recommendations
Fasting: The gist of fasting is relatively simple. You just don’t eat for a while. While you are fasting, you can still drink calorie free beverages, like water, coffee, tea, and diet soda. This is technically a water fast.
I’m not aware of any research that shows health benefits from dry fasting, that you do not also get from water fasting. Unless you have a specific reason to do one, it’s probably best to stick with the water fast.
Some people shouldn’t fast, because of health issues they have. If you’re one of those people, odds are, you probably already know. There is some information that explains how people with type 2 diabetes might be able to fast safely. I don’t know how good that information is, but I do know that some people have been using fasting to reverse type 2 diabetes, so somebody’s doing it.
Side note for feedees who might be considering weight loss surgery. Weight loss surgery is basically surgically enforced fasting. Before you make the decision to take on the risks that come with weight loss surgery, I ask that you consider checking out Dr. Jason Feung’s blog series on fasting. Your decisions are your own, but I think you’ll find following his advice to be a safer alternative to surgery.
When I first started with intermittent fasting I started with 24 hour fasts, and it took a few weekly fasts, before I started to adapt to them. If I had to do it over, I’d start with one 12 - 16 hour fast each week, before moving on to one 24 - 36 hour fast each week.
If you’re actively gaining, I’d break the fast with a huge meal, and eat well past the point of fullness. When I did this, it also started to take more food to satisfy my hunger on non-fasting days, so it’s probably think it’s safe to say that it stretches the stomach.
If you’re not actively gaining, just break the fast with a regular sized meal.
If you choose to do an extended fast, odds are, you’ll need to break it differently. I’m not going into extended fasts here, because there’s a bit of added risk, and I don’t think they’d be particularly useful for feedees. I have the vague sense that an annual or semi-annual 3 - 7 day water fast might be beneficial, but I haven’t looked into it closely, yet.
I don’t know what the best overall fasting-feasting strategy might be, but one 24 - 36 hour fast per week, followed by deliberate overeating worked especially well. Sumo wrestlers fast for about 12 - 16 hours, and break their fast with a feast of sorts; and they seem to do a good job of putting on weight.
In my own experiments, I’ve found it difficult to do more than one 24 hour fast in a week, but easy to do multiple 12 - 16 hour fasts throughout the week.
I also found that the fasts were easier, when I started out by skipping dinner, but if I didn’t follow those fasts up with some deliberate overeating, I’d lose weight; without meaning to do so.
Diet: There isn’t much point in providing much detailed information on diet.
Here’s what I think is probably the most important to know. The foods that are usually considered to be the bad foods, also tend to be a cause of inflammation.
Many of the usual “good” food recommendations like fruit, vegetables, and foods high in Omega-3 fatty acids are also considered to be anti-inflammatory foods.
I’ve done all this writing and still haven’t gotten to why inflammation is bad. Inflammation isn’t bad, exactly, but chronic inflammation is know to contribute to the usual health bad guys, like diabetes, heart disease, and cancer. [ link ]
Overall, my feedee recommendation here, is to be on the see food diet, or the all-you-can-eat diet (eat as much as you can, when you can), but to also include some fruits, nuts, and vegetables in the mix.
Exercise: I already went over the usual blah blah about seeing a doctor in my previous post, so here I’ll just say, when in doubt, see a doctor.
This section is relatively short, because I’ve already covered the whats and the whys of exercise. I also gave a few exercise ideas in my previous post.
My suggestion is to get at least 20 - 40 minutes of light physical activity 3 - 6 days out of the week. Do whatever kinds of exercises that you like the most, or dislike the least.
If you’ve been inactive for a long time, don’t start with 20 or 40 minutes. Start with 2 minutes, and then add 1 minute to the length of your (dare I say?) workout, each time you exercise. Also, don’t start with 6 days each week. Start with 3 days. Your body will need a bit of time to recover from each workout.
If exercising causes you to feel pain, don’t do the thing that causes you pain.
If regular exercise is too much for you, and it might be if you are especially heavy, I included multiple links to seated and in-bed exercise ideas in the exercise section of my previous post. Have a look at them, and try what works best for you.
If you are able to do so, I think it’ll also be worth your while to try standing for about 2 minutes every hour or so, during your waking yours. Just working against gravity for short periods of time will provide some health benefit.
Supplements For Feedees:
I’ve had an interest in herbal and nutritional medicine for a long time. There’s a lot I could say in the subject. However, this series is already about three posts longer than I originally intended to make it, so I’ll try to keep it specific feedee health.
Feedee or no, it is my believe that a good multi-vitamin and mineral supplement, and a good vitamin C supplement should be the foundation of any supplement program. Just because feedees might eat a lot, doesn’t mean we get all the nutrients we need.
The inexpensive, over-the-counter vitamin tablets that you can find in most stores get a bad rap, because they don’t digest well. However, they usually digest better, when they’re taken with a meal.
The main focus here will be insulin sensitivity, inflammation, and heart health. There are herbs that help to stimulate the appetite, but in all my searching, I have yet to find a food or an herbal or nutritional supplement that has been shown to help store the calories you’ve already eaten; as fat; more efficiently and effectively.
However, if somebody does know of something other than diabetes medications, that does this, I’d love to hear about it.
Considering that we already know that insulin causes you to store fat, and insulin sensitive fat is known to help you get fatter, I suspect that nutrients that help with insulin sensitivity will do the trick.
The typical recommendation for increasing insulin sensitivity, is to lose weight. Since weight loss is kind of the opposite of what most feedees want, it’s a good thing we have some other options?
Supplementing with some vitamins and minerals, like magnesium [ more ], chromium, and alpha lipoic acid [ more ], and vitamin D [ more ] appears to help with insulin sensitivity,
[Article: Vitamin deficiencies in people with diabetes. ]
[ Article: Dr. Whitaker - The Best Supplements for diabetes. ]
A few more:
Garlic [link] - Heart health. Cholesterol. Infections. Anticoagulant.
Turmeric - [link] Anti-inflammatory. Insulin resistance. Anticoagulant.
Fish Oil - [link] Anti-inflammatory. Heart health. Lowers triglycerides. Anticoagulant.
Cinnamon - [link] Insulin sensitivity.
Milk Thistle - [link] Supports liver and galbladder.
MSM - [link] [link] Joint pain. Inflammation. May be important for mobility.
I hope you find some of what I’ve written in these post useful. When it comes to trying to be healthy, as feedees, we’re kind of on our own. Most of those who are open to helping fatties be healthier, tend to do so by telling us to lose weight; and I’m not aware of any health resources that try to tell people how to be healthier, while getting fatter. (At least, until now, though I don’t feel particularly qualified for this..)
If medical researchers were to shift their focus toward how to help people stay healthier as they get fatter, it’d certainly help us, but there are a lot of non-feedist fatties who genuinely struggle with their weight; who would also benefit from such research; and there are a whole lot more of them, than there are of us.
It’d be nice to be able to simply open up a web browser, and find a multitude of feedee friendly doctors nearby, but if they exist, they’re not making themselves easy to find. (Multitude, huh? I think that’s the first I’m I’ve used that word, but it fits. ...but then again, maybe I’m just using big words to make people think I’m more photosynthesis.)
If nothing else, I’d like to think that what I’ve put together, in this series of posts, is the start of something that brings better health and longer lives for feedees and non-feedist fatties; alike.
Part I
I have a multi-part post about things I believe a feedee can do to improve their health; even while deliberately gaining. Some are probably obvious. Others are probably not so obvious.
It’ll probably be a few days or more, before I’m ready to start posting them, but when I am, I’ll post them here.
It’s taking longer than expected to put together. I guess I had more to say on the subject that I realized. It’s not final, but it’s looking like it’s probably going to be a 5 part series.
I have a multi-part post about things I believe a feedee can do to improve their health; even while deliberately gaining. Some are probably obvious. Others are probably not so obvious.
It’ll probably be a few days or more, before I’m ready to start posting them, but when I am, I’ll post them here.
About Me
My other blog has a very specific theme, that I would like to keep intact, but I wanted a place to post some of my feedist thoughts, creations, and pics that might not fit into that theme. I also wanted a place where I could post these things, where they wouldn’t get lost, among all the stuff I reblog. Here, I plan to post my own thoughts, my own research on the science about weight gain, my own creations, and... probably... my own pics. My main blog is mostly reblogged pics that show fat men, pics of and women admiring fat men. I didn’t originally set out to create anything like it, and there is a bit of a story behind it. When I joined tumblr, I wasn’t setting out to create a tumblr blog. I was joining to access the blog managed by a couple of female feeders, after seeing a link posted to it; elsewhere. After joining, I found myself following the pic blogs of bbws, ssbbws, and female feedees. Sure, I’m attracted to fat and supersized women, but I’m also attracted to all (or at least most) other female body types too; to one degree or another. I’m not attracted the male body. I love seeing fat, and seeing female feedees, bbws, and ssbbws allows me to both enjoy the fat, and my attraction to women; which is my I’ve mostly followed bbw, ssbbw, and female feedee blogs.
(...and make no mistake, even though I like pics of supersized, female feedees, I’m attracted to skinny women too, and I want to be the one who is the feedee.) Somewhere along the way, it occurred to me that one of my own challenges as a feedee was that I couldn’t see fat men as being attractive; which made it hard for me to picture any woman loving my fat, and being turned on by making me fatter. (That is what I want, after all.) That led me to decide that it was time for me to start looking at pics of fat men, to see if I could see how some women might find them attractive. What I was really looking for, was proof that some women were legitimately attracted to fat men (at least in part), because of their fat. What I found was a whole lot of content that showed gay men being legitimately attracted to fat men, and having feeder tendencies. I found very little of anything, showing women being attracted to fat men, but I did find some stuff. In time, I found more. It was then, that I decided that I should create something that I thought female feeders and ffa’s, who are attracted to men, might enjoy. It also helps sometimes, to remind myself that there are women who would be attracted to a guy like me.
I sometimes notice when I catch the eye of a woman; in passing, but it’s not the same as knowing she’d be turned on by fat, and would be sexually driven to make me fatter.
...and being able to imagine a woman being turned on by my fat, and having powerful sexual drive to make me fatter is what “does it“ for me the most. There’s a lot more to relationships than sex, but goddamnit, sometimes I want a woman who is so turned on at the thought of making me fatter, that she feels the need to masturbate as she watches me stuff myself.
I’m not quite sure where I stand on relationships, right now though, for a number of reasons, but what I know for sure, is that I don’t want a committed relationship with a woman who isn’t an FFA or a feeder.