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lulasissy-deactivated20260208

Femboi-love💕

its-lexi-things

So sexy…💋🍆💋

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Ararity

mudwerks

(via Trade Marks of Caution, 1951 – Present & Correct)

chieffoxengineer10

oldshowbiz

Jazz music started doing acid long before the rock stars of the late sixties. Dizzy Gilespie, Charles Mingus, John Coltrane, and Thelonious Monk were all trying psychedelics long before Janis Joplin, Jimi Hendrix, or Donovan came along.

Psychedelics in jazz came from a number of sources. Zoot Sims picked up his LSD from reverb virtuoso Sandy Bull. Philosopher Gerald Heard was the main supplier for the West Coast Jazz scene. Michael Hollingshead provided LSD to Maynard Ferguson. Timothy Leary provided psilocybin to Dizzy Gillespie. Allen Ginsberg provided Thelonious Monk with both psilocybin and LSD.

“A few days later Ginsberg dropped by Monk’s apartment to check on the results,” said Martin A. Lee. “Monk peered out from behind a crack in the door, smiled, and asked if he had anything stronger. Ginsberg also turned on Dizzy Gillespie, who was evidently quite pleased by the gesture. ‘Oh yeah,’ he laughed, ‘antyhing that gets you high.’ In a sense it was Ginsberg’s way of returning historical favor; the jazz musicians had given marijuana to the beats, and now the beats were turning the jazz cats on to psychedelics. Word of the new drugs spread quickly through the jazz scene, and numerous musicians, including many of the preeminent players in the field, experimented with psychedelics in the early 1960s. John Coltrane….”[iii]

Tenor sax player Charlie Rouse and Thelonious Monk were playing at the Jazz Workshop in Boston when Timothy Leary paid them his first visit in 1961. He came backstage and provided them both with psilocybin pills. Word began to spread to other jazz musicians. When you’re in Boston - try this guy.

Monk followed up in August 1962, asking for LSD. Leary referred him to Ginsberg while Monk was booked at the Village Gate. His opening act was Hugh Romney, an acid head comedian who later became famous at Woodstock as Wavy Gravy.

Trumpet legend Maynard Ferguson and his wife Flo crashed at Timothy’s Leary’s home after a Cambridge gig in December 1961. Leary had only done psilocybin up to that point, never LSD. Michael Hollingshead administered LSD to Maynard and Flo that evening and as it kicked in, they encouraged Leary to try it. Maynard Ferguson shouted, “Yeah, really, Tim, it really gets you there – wow – it’s really happening, man.” [iv] The beaming, positive, aura of the two jazz musicians convinced Leary to try LSD for the very first time.

Charles Mingus came by Leary’s compound the next time he was in town. It was an exciting time for Jazz and LSD. According to the book Storming Heaven, “Maynard Ferguson [would be] standing on the rooftop sending long trumpet rills snaking out over the gardens, in which another jazz legend, Charlie Mingus, could sometimes be found pruning the rosebushes.”[ii] [iii]

John Coltrane became spiritual after his first acid doses in 1965-66. He kicked his crippling opiate habit after LSD reset his brain. With a new lease on life, Coltrane started performing religious-themed jazz music and it is said that his holy LSD experiences were the basis for his next big album – A Love Supreme.

“From 1965 to his death in 1967, Coltrane was certainly performing under the influence of LSD,” writes Tony Whyton in his pretentious book Beyond Love Supreme. “By using LSD, Coltrane moved closer to Eastern mysticism and embraced the spirit of experimentation at the time … With the use of a drug such as LSD, Coltrane could be carried beyond his physical being in quest to become disembodied, fusing his sound with nature.”[iii]

Donald Garrett, a jazz bassist and clarinetist, was among those who did LSD with John Coltrane. It was autumn 1965 in Seattle. “There, they had taken LSD before recording an album.” wrote J.C. Thomas. “The pervasive influence of ingesting acid may have produced the eerie, mystical vibrations that emanated from their recording of Om, which included, in addition to some of the freest and strangest music Coltrane ever recorded, the chanting of selected verses from Bhagavad-Gita. And when Trane returned from his LSD trip, he said, as if quoting a Sufi safe, ‘I perceived the interrelationship of all life forms.”[iv]

Jazz writer and critic Larry Hickock said, “I was on LSD the first time I heard Coltrane, and the record was Ascension. I was nineteen, had a full tab of acid under my belt, and heard this overpowering musical force filling my head to overflowing … It affected me so much I stopped getting high … And when I heard Spiritual, I knew then what Coltrane really was – a spirit.”[viii]

The combination of John Coltrane and LSD inspired a new generation of musicians who felt the undercurrent of psychedelic awareness in his music. Grace Slick of the Jefferson Airplane described the LP Ascension as “Coltrane’s acid trip.” [v]

Carlos Santana was high on psychedelics during his legendary Woodstock performance. Years later he said, “I’m comfortable with my existence and the things that I did learn from mescaline and LSD. I don’t regret one trip. I learned a lot. But the thing that makes the most sense is John Coltrane’s music.” [vii]

Devoted acid heads like The Byrds were overwhelmed by Coltrane’s sound. “It felt like a white hot poker searing through my chest,” said Roger McGuinn. “It cut deep into me and it was a little painful. But I loved it. It opened up some areas in my heart and head that I hadn’t know about.”[ix] The Byrds said their hit song Eight Miles High was “Coltraine-inspired.” McGuinn explains, “The first break [of Eight Miles High] is a direct quote from a Coltrane phrase and throughout the rest of the song, we try to emulate the scales and modes that Trane was using. Especially his spiritual feeling, which got me into transcendental mediation not long afterwards.”[xi]

Record mogul Richard Bock, the head of Pacific Jazz Records, was the first record executive to sign The Byrds. Pacific Jazz was the primary label for the West Coast Jazz scene. Bock was an early psychedelic adherent thanks to his two closest friends in the early 1950s - philosopher Gerald Heard and Doors of Perception author Aldous Huxley.

Heard was an eccentric Englishman who ran an artist’s commune in Southern California. He became the primary source of LSD for both Aldous Huxley, horn player Paul Desmond, Alcoholics Anonymous founder Bill Wilson, and jazz legend Dave Brubeck. Pacific Jazz released a three-LP spoken word set called Gerald Heard Re-Birth at the end of the 1950s. The album was based on The Tibetan Book of the Dead and the psychedelic concept of death and re-birth. The liner notes explained: “This is a series of reflections and meditations on those spontaneous cryings out of men who were searching and yearning for the answers to the eternal questions of Where Am I? What Am I? Who Am I? – which illustrated the development of the human spirit, the growth of man’s consciousness.”

By the end of the 1950s, Bock, Heard, Huxley, and novelist Christopher Isherwood were doing LSD on a regular basis. Their pedigree and reputation was top notch and they were able to conduct salons of accomplished people who wanted to try acid. Among them were Tonight Show host and jazz ally Steve Allen [ii], legendary filmmaker John Huston [iii], writer Terry Southern, and the publishers of Time Magazine - Henry and Clare Booth Luce. “Oh, sure, we all took acid,” said Booth Luce. “It was a creative group – my husband and I and Huxley and Isherwood.”

Richard Bock’s Pacific Jazz Records was also the first company to put a record on the market that referenced LSD. The instrumental surf single - LSD-25 by The Gamblers. Released in 1960, it was an early birth pang of the 1960s surf music craze.

Surfing and LSD were intertwined in the early 1960s. Musician Bobby Jameson, who was Diane Linkletter’s acid dealer, got his start playing instrumental surf music. Surfing organizations like The Brotherhood of Eternal Love distributed thousands of hits of acid across Southern California, smuggled inside surf boards from Hawaii. And some of Bock’s West Coast Jazz artists like Bud Shank recorded soundtracks for independent surfing-footage movies.

Richard Bock’s friendship with Aldous Huxley would come full circle. Bock saw potential in a rock band called Rick and the Ravens and put them in his recording studio. He seldom pressed rock acts on his label, it was usually just jazz music or the LSD-using comedian Lord Buckley. But these guys seemed different. Before too long, The Ravens renamed themselves after Aldous Huxley’s psychedelic book The Doors of Perceptions. They became known as The Doors.[xii]

Listen to the LSP inspired jazz album that vibist Emil Richards record in 1967 here.

(This is a chapter about Jazz and LSD from an unpublished manuscript)

trashypics

mandatory-feminization

Feminizing Hormone Regimens

I receive frequent inquiries from transgender persons who want my advice concerning feminizing hormone regimens. Naturally, I believe that taking hormones under medical supervision is by far the best and safest course.

But I also believe that transsexual women who decide for whatever reason to take hormones without medical supervision ought to have as much information as possible to guide them. Here are my thoughts:

Estrogen is the most important part of any feminizing regimen. Some typical initial estrogen dosages for preoperative transsexual women who have not undergone SRS or orchiectomy (castration) are as follows:

estradiol (e.g., Estrace® or Estrofem®), 6-8 mg daily; OR

estradiol valerate (e.g., Progynova®), 6-8 mg daily; OR

conjugated equine estrogens (e.g., Premarin®), 5 mg daily; OR

ethinyl estradiol (e.g., Estinyl®), 100 mcg (0.1 mg) daily (NOT RECOMMENDED); OR

Transdermal estrogen:

estradiol (e.g., Vivelle-Dot®, Estraderm®, Climara®, etc.), two 0.1 mg patches, applied simultaneously, changed per manufacturer’s recommended schedule (see note below); OR

Injectable (intramuscular) estrogen:

estradiol valerate (e.g., Delestrogen®), 20 mg IM every two weeks.

Occasionally half the suggested dosage may be sufficient. Sometimes the dosage will need to be increased, rarely even doubled. Beyond a certain point, larger dosages will not increase tissue response, but will only cause more side effects.

Oral estrogens are most commonly used, and are typically very satisfactory. Among the oral preparations, I prefer estradiol (or estradiol valerate, which is virtually identical). Estradiol is very inexpensive, and has low hepatic toxicity. Most clinical laboratories can perform estradiol blood levels; it is more difficult to obtain meaningful measurements of blood levels with conjugated equine estrogen or with ethinyl estradiol. Estradiol is also produced synthetically, without cruelty to animals. This is not the case with conjugated equine estrogen (Premarin®), which is prepared from the urine of pregnant mares.

Estradiol tablets can be taken sublingually (placed under the tongue to dissolve) instead of being swallowed. This may reduce possible liver toxicity, because with sublingual administration, much of the medication is absorbed directly into the blood stream, rather than being metabolized by the liver after first passing through the digestive tract. Less metabolism is also likely to result in higher levels of estradiol itself, and lower levels of its less-active metabolites, estrone and estriol. Micronized estradiol tablets are specifically designed for either oral or sublingual use and dissolve quickly under the tongue without an unpleasant taste.

Premarin® is by far the most expensive oral preparation. One of its few advantages is its relative potency, which is notably higher than estradiol on a milligram-per-milligram basis. This is because some of the equine estrogens in Premarin, especially equilin, have higher biologic potency than the estrogens normally found in humans.

Ethinyl estradiol is a chemically-modified form of natural estradiol. The ethinyl substitution results in a longer duration of action, and greatly increased potency. Consequently, typical milligram dosages of ethinyl estradiol are about one-fiftieth of typical milligram doses of estradiol. This is a preparation I do not recommend, due to its association with thromboembolic complications; see Toorians et al. (2003). Venous thrombosis and changes of hemostatic variables during cross-sex hormone treatment in transsexual people. Journal of Clinical Endocrinology and Metabolism, 88, 5723-5729.

I think that taking 81 mg of aspirin daily is a good precaution for persons taking oral estrogens, assuming no contraindication to aspirin exists.

Transdermal estrogen causes less clotting tendency than oral estrogen, which is possibly important to some patients. However, transdermal preparations are more expensive, and skin reactions to the adhesives employed are not uncommon. I recommend transdermal estrogen for most patients over the age of 40, to patients with risk factors such as cigarette smoking, and to patients with a personal or family history of cardiovascular disease. Transdermal patches should be changed according to the manufacturer’s recommended schedule: twice weekly for most patches, weekly for Climara®.

Injectable estrogen may cause less clotting tendency than oral estrogen and it is less expensive than transdermal estrogen. However, it requires the use of needles and syringes, and the ability to perform injections; it has a greater tendency to increase serum prolactin levels; and it is often associated with inadvertent or deliberate overdosage. Contrary to the belief of many consumers, there is no credible evidence that injectable estradiol produces superior feminization. I do not recommend the use of injectable estrogen and I no longer prescribe it in my practice.

If you have access to laboratory testing, a serum estradiol level of about 125-200 pg/ml – about one-third to one-half the normal female mid-cycle peak – is often considered ideal, at least for the first two years or so of feminizing therapy. It is not necessary or desirable to “cycle” estrogen, or any other medication, in an attempt to mimic the normal female menstrual cycle. Besides providing estrogen, a hormone regimen should also reduce testosterone to normal female levels. This usually requires adding an anti-androgen.

In persons who have not had an orchiectomy, reducing testosterone levels is also a concern. Although the desired reduction in testosterone can theoretically be accomplished with estrogen alone, the dosage required is usually in excess of what is needed for feminization. Adding an anti-androgen allows lower dosages of estrogen to be used; this is usually highly desirable. Typical dosages of anti-androgens are as follows:

spironolactone (e.g., Aldactone®), 100-300 mg daily in divided doses; OR

cyproterone acetate (e.g., Androcur®), 100-150 mg daily.

Sometimes 100 mg of spironolactone may be sufficient, but 200–300 mg is a more typical dose. The Vancouver group uses up to 600 mg daily, apparently without problems. Spironolactone is fairly inexpensive, is readily available, and is usually quite well tolerated. In my opinion, it should be regarded as the anti-androgen of choice for most patients. Cyproterone is actually a progestin (see below), but it is used primarily for its anti-androgenic effects and only secondarily for its progestational effects. It is not available in the US, but is popular elsewhere. One disadvantage of cyproterone is that it counteracts some of the desirable effects of estrogen on blood lipids. Cyproterone, when used in combination with estrogen, may also share some of the increased risks associated with the synthetic progestin medroxyprogesterone when so used (see below).

If you have access to laboratory testing, a serum testosterone level within the normal female range – about 5-85 ng/dl for total testosterone, or 0.1–2.2 pg/ml for free testosterone – is usually considered ideal. Within the female normal range, lower numbers are not necessarily better. Progestins (progesterone and synthetics) are sometimes added to a hormone regimen. I consider them unnecessary for most patients.

Progestins are most often given in an attempt to increase breast development. Based on limited anecdotal evidence, I think that improved breast development sometimes can occur, but that the effects are usually not very significant. Progestins can also inhibit testosterone production, and are sometimes used for this purpose. I consider progesterone and other progestins to be unnecessary for most patients, and I prescribe them only rarely. If you decide to take them, here are some typical dosages:

medroxyprogesterone (e.g., Provera®), 5-10 mg daily; OR

micronized progesterone (e.g., Prometrium®), 100 mg twice daily; OR

Injectable (intramuscular) progestins:

medroxyprogesterone (e.g., Depo-Provera®), 50 mg every two weeks; OR

progesterone in oil, 50 mg every two weeks.

Oral medroxyprogesterone, the most commonly used product, is very inexpensive, but it has the disadvantage of counteracting some of the beneficial effects of estrogen on blood lipids. Some people find that it causes depression or mental irritability. The recently published Women’s Health Initiative study has also documented an increased incidence of adverse complications in women taking medroxyprogesterone in combination with conjugated estrogens for hormone replacement; this increased incidence of adverse complications was not found with conjugated estrogens alone. Micronized progesterone is a reasonable alternative in those who want to take a progestin. It does not counteract the beneficial effects of estrogen on blood lipids. But micronized progesterone is more expensive, and is often harder to obtain. When taken by mouth, it is partially metabolized to 5-alpha and 5-beta pregnenolone; these metabolites can act as natural tranquilizers, and may promote sleep. This effect may be desirable in patients who suffer from anxiety or insomnia. After orchiectomy (castration) or SRS, dosages can be reduced. Following SRS, anti-androgens can be discontinued, and estrogen dosage can usually be decreased to one-half or one-quarter of the pre-op dosage, i.e.:

estradiol (e.g., Estrace® or Estrofem®), 1-2 mg daily; OR

estradiol valerate (e.g., Progynova®), 1-2 mg daily; OR

conjugated equine estrogens (e.g., Premarin®), 0.625-1.25 mg daily; OR

ethinyl estradiol (e.g., Estinyl®), 20-50 mcg (0.02-0.05 mg) daily; OR

Transdermal estrogen:

estradiol (e.g., Climara®, Estraderm®, or equivalent), 0.05-0.1 mg, changed per manufacturer’s recommended schedule; OR

Injectable (intramuscular) estrogen:

estradiol valerate (e.g., Delestrogen®), 5-10 mg IM every two weeks.

(Injectable estradiol is rarely used after orchiectomy or SRS because the doses required are so small.)