#surprisednotsurprised

[sullythebarber]

#fsu #inthenewsagain #surprisednotsurprised #rightcross #connects #pitiful #embarrassing #toughguy #jailhouserock #nflready #fitrightin #gimmemydrink #now #notnice #notniceatall #seminole #prosecutethispunk #turntup #mrfootball #angermanagement #growupbro #floridastate #fsuofficialsstandup #500dollarbond #joke

[myradish]

A groundbreaking 2006 study found "striking" relationships between six POPs and diabetes in U.S. adults exposed to normal levels of POPs. The higher the levels of these POPs, the higher the prevalence of diabetes. In the highest exposure group, the risk of diabetes was 37.7 times higher than in the people with the lowest levels of exposure. That is a lot-- far higher than any other study I have ever seen. The POPs included the dioxins HpCDD and OCDD, DDE, PCB-153, oxychlordane, and trans-nonachlor, with the latter three showing the most significant relationships. (Oxychlordane and trans-nonachlor result from the use of the organochlorine pesticide chlordane) (Lee et al. 2006). Surprisingly, this study found that obesity did not increase the risk of type 2 diabetes if those people had very low levels of POPs in their bodies. In an editorial in The Lancet, Porta (2006) writes, "This finding would imply that virtually all the risk of diabetes conferred by obesity is attributable to persistent organic pollutants, and that obesity is only a vehicle for such chemicals. This possibility is shocking."

http://www.diabetesandenvironment.org/home/contam/pops

You know how it's a thing that being fat correlates with type 2 diabetes?

Apparently, this is only because POPs accumulate in the fatty tissues:

" obesity did not increase the risk of type 2 diabetes if those people had very low levels of POPs in their bodies "

Read this part again:

"This finding would imply that virtually all the risk of diabetes conferred by obesity is attributable to persistent organic pollutants, and that obesity is only a vehicle for such chemicals."

This means that whether or not you are fat, it is exposure to POPs that is the major risk factor for type 2 diabetes, not diet or exercise. 

What really strikes me is this part:"A study found that higher levels of hexachlorobenzene (HCB), some PCBs, and DDE (but not mirex) were associated with diabetes in adult Native Americans of the Mohawk Nation. "

The Kahnawake Schools Diabetes Prevention Program (Kahnawake is a Mohawk reservation) has been running since 1994. I'm going to talk to the people involved about these two studies to see what they think.

Overall, people of colour and indigenous peoples have higher rates of type 2 diabetes. Generally the reasoning behind this is, bluntly put, that we are poor, fat, and lazy.

However, people of colour and indigenous peoples also have higher rates of exposure to toxins such as POPs.

So this would make a shit-ton of sense.

But the first study came out in 2006, and yet all you hear about diabetes prevention is the same old individual-blaming BS. 

Community endeavors like the KSDPP are rare, and if they are also based on misinformation, then how is anybody supposed to help prevent diabetes in their communities?