My very last overnight shift of clinics and I’m fighting foals and spying on mares waiting to see if they go into labor. Also my allergies are starting to get to me. How do horse people do it?
Foals are smaller and weigh less, but damn do those little hooves hurt like hell. It is also still 75 kilos going full force on your foot, because the other three legs it has have not yet properly learned how to keep Standing™
"They were designed to run really really fast until a wolf or a sabertooth tiger or something eats them...or eats their slower friend. Then they go back to eating."
~Equine Professor regarding the evolution of the horse
A study led by Dr. Sue Dyson found signs of lameness in 73% of horses riders identified as sound. Also, nearly 50% of the horses' saddles sh
TheHorse.com | 30 Sept, 2020
Learning Not to Accept Signs of Pain as Normal
The fact that riders don’t recognize signs of pain or lameness in their horses is problematic, said Dyson. “I am always disappointed when you have a sample of volunteer horse/rider combinations in which the horse is considered clinically sound, and you find that unfortunately it’s not,” she said. “And we see a high proportion of those.”
This doesn’t mean the riders aren’t paying attention to their horses. Rather, it’s probably reflective of the riders’ past experiences with horses and lack of training in recognizing pain-related behavior. Specifically, many people’s first experiences with horses are in riding schools, where the horses tend to have subtle pain due to subclinical lameness, suboptimal saddle fit, and carrying unbalanced and/or unskilled riders, Dyson explained.
“We believe that many riders learn to ride on riding school horses which exhibit these signs, so that there is a generalized acceptance that these behaviors are normal for horses,” she stated. “There is an urgent need for education of riders and trainers at all levels to recognize that demonstration of eight or more behaviors of the RHpE is highly likely to reflect musculoskeletal pain.”
The study: Application of a Ridden Horse Pain Ethogram and Its Relationship with Gait in a Convenience Sample of 60 Riding Horses
Background: Warmblood fragile foal syndrome (WFFS) is a fatal, inherited connective tissue disorder in horses that shares similarities with
7 January, 2021
The research: An international team of scientists from Austria, Germany, Poland, and the United States, led by the UC Davis Veterinary Genetics Laboratory Director Dr. Rebecca Bellone, investigated the distribution of the WFFS allele across 38 different horse breeds in Europe and the USA, screening 4081 horses. They also probed the hypothesis that the WFFS allele originated in the Arabian breed. To assess the allele's prevalence in modern Arabians, they isolated DNA from 302 Arabians from diverse lines. To investigate whether the Arabian stallion Bairactar Or. Ar. (1813) was the founder of the allele, researchers isolated DNA from a museum specimen of his skeletal remains and analyzed it for the WFFS variant.
The results: 200 horses (4.9%) representing 21 breeds carried the WFFS allele. Most of these breeds were warmbloods; however, a few non-warmblood breeds also carried the allele. The non-warmblood exceptions include the Thoroughbred (carried by 17 out of 716 horses), the Knabstrupper (3/46), the Haflinger (2/48), and the American Sport Pony (1/12).
The WFFS allele was detected in 17 out of the 19 tested warmblood breeds, with the highest carrier frequency observed in Hanoverians and Danish Warmbloods (both approximately 17.3%). The allele was not detected in two warmblood breeds, the Swedish Warmblood and Zangersheide Warmblood, but sample sizes for both breeds in this study were small (n = 16 and n = 10, respectively). The average WFFS carrier frequency across all warmbloods tested was 11%.
The WFFS allele was not detected in any of the 302 Arabians tested, nor was it detected in the DNA sample isolated from the historical sire Bairactar Or. Ar., suggesting that an Arabian origin of the allele is unlikely. The origin of the WFFS allele remains unknown.
For more on this study see: Distribution of the Warmblood Fragile Foal Syndrome Type 1 Mutation (PLOD1 c.2032G>A) in Different Horse Breeds from Europe and the United States