Andeluuu

Watch films actively, read voraciously, and write fearlessly. I am a scientist, but man do I love me some film. On top of being a scientist who actually enjoys being in a lab, most of the time, I've come to love cinema, and writing because I like to hear myself think.

[writeme-andeluuu]

I've missed like two weeks in a row, that is bonkers. I need a better schefuling app, or I just need to get my stuff together. I am trying to get back to the routine, in the time that had passed I did work on investigating wether Anti-Malarial rememdies of the past, drinks that are now known as tonic water, were effective remedies for Malarial infections. As it turned out, no, no they weren't.

The Weekly Report - Trying to get back on a schedule.

[theotherway]

“Every reader, as he reads, is actually the reader of himself. The writer’s work is only a kind of optical instrument he provides the reader so he can discern what he might never have seen in himself without this book. The reader’s recognition in himself of what the book says is the proof of the book’s truth.”

— Marcel Proust, Time Regained (via mesogeios)

[writeme-andeluuu]

Rachel Sennot as Rosie Shuster, Saturday Night (2024)

[sanriovault]

banana split ♡︎

[frommylimitedtravels]

Hoh stuff - May, 2025

[expressions-of-nature]

by hajime3776

[travellingwiththedead]

Leishmaniasis of the skin

[onlytiktoks]

Chagas disease, or deadly kissing bug disease, has spread in the U.S. Here's what to know.

Chagas disease​ is already endemic to 21 countries in the Americas, and growing evidence of the parasite is challenging the non-endemic labe

About Chagas Disease

Chagas disease is caused by a parasite. It is passed through the droppings of the triatomine bug.

[biologist4ever]

A Look Inside a Brain Cancer Cell

This isn't just a pretty picture; it's a map of how a brain cancer cell thrives.

The luminous cyan web is the actin cytoskeleton. It's the cell's scaffolding and its engine, constantly remodeling to push the cell forward through the brain's delicate tissue.

The fiery red network is the endoplasmic reticulum (ER). Think of it as the cell's intensive production and logistics hub, synthesizing proteins and managing the intense stress of rapid, chaotic growth.

When you see them together like this, you're witnessing the fundamental machinery of invasion. The cell isn't just growing; it's strategically rewiring itself to survive and spread.

[mindblowingscience]

Pancreatic cancer mRNA vaccine shows lasting results in an early trial

Scientists caution that more research is needed, but nearly all of the patients who responded to the personalized vaccine are still alive si

Donna Gustafson had a harder time than usual shaking off the jet lag from her 22-hour journey from Florida to Australia. Two days into her trip, her skin took on the yellow hue of jaundice. Gustafson, who is now 72 and lives in Delray Beach, Florida, went to the emergency room for fluids, thinking she was dehydrated. In a surreal moment, the Australian doctors instead told Gustafson that she had pancreatic cancer. “They were very adamant about it,” Gustafson said. “This is absolutely pancreatic cancer.”

Continue Reading.

[haporiumblog]

Ever wonder how one of the deadliest cancers just… slips past our body’s defenses? Turns out, pancreatic tumors are masters of disguise, and they’re using a surprisingly sweet trick. 🍬 New research is pulling back the curtain on their sugary shield, and it’s a game-changer for treatment.

The Sugar-Coated Lie

Pancreatic cancer cells are like stealth fighters. They coat themselves in a dense layer of sugar molecules called sialic acids. This isn’t just decoration—it’s a full-body camouflage suit against our immune system.

Think of your immune cells (the T-cells) as security guards. They’re supposed to recognize and destroy invaders. But these tumor cells are so thoroughly “sugar-coated” that they appear harmless, flying under the radar.

Hacking the Immune “Off” Switch

Here’s the real kicker: it’s not just about hiding. This 2026 discovery found a specific protein, an integrin, that gets absolutely covered in these sialic acids (becoming “hypersialylated”).

This sugary integrin acts like a molecular handshake with certain immune cells.

That handshake doesn’t signal “attack.” Instead, it delivers a powerful “stand down” order.

The immune system is effectively deactivated at the tumor’s doorstep, letting cancer grow unchecked.

A Sticky New Target for Therapy

This breakthrough in glyco-immunology flips the script. We’ve been trying to boost the immune system to fight better. Now, we can also think about stripping away the tumor’s disguise.

The goal? Develop drugs that:

Block that sugary integrin handshake.

Or, strip off the sialic acid shield entirely.

Once the tumor’s sugary armor is gone, our immune cells can finally see it for the threat it is and attack. This isn’t

#Immunology #MedicalResearch #CancerTreatment #CancerResearch #Immunotherapy

🔗 Read more on Haporium

[bpod-bpod]

Topical Treatment

From dengue to Zika, mosquito-borne viruses are a significant global problem, likely to grow in the face of climate change. Many of these viruses co-occur and initially cause similar symptoms, making early diagnosis difficult, but recent research suggests that multiple viruses could be tackled in the same way, by boosting immune defences in the skin. Experiments on both mice and human skin samples suggest that quickly applying a skin cream containing the anti-viral imiquimod, currently used to treat basal cell skin cancer, on mosquito bites can reduce the extent of infection by several viruses. Pictured is a section from a mouse’s brain, infected by the mosquito-borne Semliki Forest virus (in pink); imiquimod application reduced viral replication in the skin and its spread to other tissues, significantly increasing survival. Subject to testing for safe use in this context, imiquimod-containing creams could potentially provide some protection against a range of mosquito-borne viruses.

Written by Emmanuelle Briolat

Image from work by Steven R. Bryden and colleagues

Virus Host Interaction Team, Leeds Institute of Medical Research, School of Medicine, Faculty of Medicine and Health, University of Leeds, Leeds, UK

Image copyright held by the original authors

Research published in Science Translational Medicine, January 2020

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[thatmohstech]

Nodular basal cell carcinoma in a first stage Mohs layer

[dazzlingyouwithmywits]

“They have no power to improve their lives, but they have the power to make others... miserable.”

-Octavia E. Butler

[hexgirlhexes]

“You saved me.”

“You saved me.”

“You saved me.”

The difference.

[petaltexturedskies]

Margaret Atwood, Cat's Eye

[bpod-bpod]

Cancer Moves

Cancer cells can invade tissues by what were thought to be two ways – one that activates enzymes to break down the tissue ahead and the other that pushes through by mechanical might (amoeboid movement). Now by studying cancer cell assemblies called spheroids, it's revealed that in fact the two modes while distinct both combine active tunnelling with enzyme breakdown by matrix-degradative metalloproteinases.

Read the published research article here

Video from work by Adam W. Olson and colleagues

Cancer Biology Department, Life Sciences Institute, University of Michigan, Ann Arbor, MI, USA

Video originally published with a Creative Commons Attribution – NonCommercial – NoDerivs (CC BY-NC-ND 4.0)

Published in Proceedings of the National Academy of Science (PNAS), April 2026

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