Agent Orange. Invented by âDow Chemicalâ and âMonsanto.â Since at least 1978, several lawsuits have been filed against these companies.In addition to its damaging environmental effects, the chemical has caused major health problems for many individuals who were exposed. Up to 4 million people in Vietnam were exposed to the defoliant, with around 1 million now suffering serious health issues. The chemical is capable of damaging genes, resulting in deformities among offspring of exposed victims. After returning home, Vietnam veterans began to suspect their ill health or the instances of their wives having miscarriages or children born with birth defects might be related to Agent Orange.Â
Batrachotoxin. Of the three so-called âpoison dartâ frogs which contain batrachotoxinâgolden poison frog, Kokoe poison frog, and neariâthe most toxic is the most recently discovered golden poison frog. This frog is so tiny that it could sit on the tip of your nail. But one frog has enough poison to kill 20 people or 2-3 elephants. As a neurotoxin it affects the nervous system. Batrachotoxin interferes with heart conduction, causing arrhythmias, extrasystoles, ventricular fibrillation and other changes which lead to cardiac arrest. No effective antidote exists for the treatment of batrachotoxin poisoning.
Botulinum toxin. The deadliest poison. The toxin can spread, blocking nerves and muscle function. In severe cases, the toxin can block nerves controlling the respiratory system or heart, resulting in death.
VX (nerve agent) Death usually occurs within 15 minutes after absorption of a fatal VX dosage. The prolonged neuromuscular blockade results in flaccid paralysis of all the muscles in the body, and it is such sustained paralysis of the diaphragm muscle that causes death by asphyxiation. An individual known to have been exposed to a nerve-agent, or who exhibits definite signs or symptoms of nerve-agent exposure are generally given the antidotes atropine as well an injected sedative/antiepileptic such as diazepam.
Ricin. Ricin is very poisonous if inhaled, injected, or ingested. It can also be poisonous if dust contacts the eyes or if it is absorbed through damaged skin. Within 2 to 5 days of exposure to ricin, effects of ricin on the central nervous system, adrenal glands, kidneys, and liver appear. The low blood volume can lead to organ failure in the pancreas, kidney, liver, and GI tract and progress to shock. Shock and organ failure are indicated by disorientation, stupor, weakness, drowsiness.
Tetrodotoxin. The toxin can enter the body of a victim by ingestion, injection, or inhalation, or through abraded skin. Symptoms typically develop within 30 minutes of ingestion, but may be delayed by up to four hours; however, if the dose is fatal, symptoms are usually present within 17 minutes of ingestion. Paresthesia of the lips and tongue is followed by developing paresthesia in the extremities, hypersalivation, sweating, headache, weakness, lethargy, incoordination, tremor, paralysis, cyanosis, aphonia, dysphagia, and seizures.
Amatoxin. Exposure to amatoxins may cause irritation of the respiratory tract, headache, dizziness, nausea, shortness of breath, coughing, insomnia, diarrhea, gastrointestinal disturbances, back pain, urinary frequency, liver and kidney damage, or death if ingested or inhaled. Â An initial (12â24 hours) period of acute symptoms is followed by a period of relative wellness that lasts for 12â24 hours. After this period, hepatic and renal failure supervene with death typically occurring from day 2 onwards. The estimated minimum lethal dose is 0.1 mg/kg or 7 mg of toxin in adults.
amandla
pixelskylines
prguitarman
