Cardiovascular complications of COVID-19
Virus infections are the most common cause of myocarditis. The previous severe acute respiratory syndrome (SARS) beta-coronavirus SARS-CoV-1 was associated with tachyarrhythmias, signs, and symptoms of heart failure.
Letâs learn about SARS-CoV-2:
COVID-19 patients who died had higher levels of troponin, myoglobin, C-reactive protein, serum ferritin, and IL-6.
This is because of the high inflammatory burden in COVID-19.
Letâs find out and understand about CVS complications by COVID-19:
Mainly, vascular inflammation, myocarditis, and cardiac arrhythmias
The possible late phenomenon of the viral respiratory infection
Commonly observed in severe cases
Strongly associated with mortality
Cardiac MRI Findings in COVID-19:
âŹïž Wall thickness with diffuse biventricular hypokinesis (especially in the apical segments)
Severe LV dysfunction (LVEF < 35%)
Circumferential pericardial effusion ( notable around the right chambers)
Short tau inversion recovery and T2-mapping sequences:
Marked biventricular myocardial interstitial edema
Diffuse late gadolinium enhancement involving the entire biventricular wall
CVS complications as an important prognostic factor:
Patients who have high troponin levels showed a higher incidence of complications such as
Lastly, what are the major risk factors for COVID-19 mortality?
Strong and independent risk factors:
the presence of comorbidities ( HTN, coronary artery disease)
cardiac injury, myocarditis, and ARDS
Inciardi RM, Lupi L, Zaccone G, et al. Cardiac Involvement in a Patient With Coronavirus Disease 2019 (COVID-19). JAMA Cardiol. Published online March 27, 2020. doi:10.1001/jamacardio.2020.1096
Madjid M, Safavi-Naeini P, Solomon SD, Vardeny O. Potential Effects of Coronaviruses on the Cardiovascular System: A Review. JAMA Cardiol. Published online March 27, 2020. doi:10.1001/jamacardio.2020.1286