#gnrh

[faxxmachine]

This is fucking monstrous. Read the whole thing and be enraged: The Dutch Protocol for Juvenile Transsexuals: Origins and Evidence by Michael Biggs

Warning: contains descriptions of chemically neutering homosexuals.

[tmitransitioning]

I have heard conflicting things about puberty blockers. I have heard they are 100% perfectly safe to use and as such any questioning kid should be allowed to have them as they have no longterm problems. But I've also heard that isn't true, though didn't have it explained why. Which is the truth?

If an afab person were to go on hormone blockers before puberty, start T at 18, and stay on T until they’re past the age at which they would have gone through menopause (I know it varies but this is hypothetical), if at that point they went off T, would they have any breast development, or would their body be past the point of producing enough hormones for that? That 17 y/o anon wondering about the puberty blockers again; are there any long term effects? What happens when I stop-- would I resume puberty? Even if I'm (possibly) past the end of puberty? Does it decrease bone density? I don't plan to go on T, and I'm agender but I'd rather just, pause my puberty for the time being because I'm pretty happy where I am now. (Though... (Tmi-ish) in the last few months I've noticed my butt is... bigger? And would def not like that to continue lmao) thanks!! Grouping all these blocker asks together to answer similar questions at once:No medicine at all is “100% safe” But in the realm of treatments, delaying puberty with blockers temporarily is low risk and high return.  The goal of delaying puberty is to hit the pause button to give the person time to determine what “direction” puberty should go in (either the one the body naturally wants to go toward, or a different one, directed by adding other hormones).   

The major risk of puberty blockers being used short-term (several months to several years) in this way is osteoporosis and short adult height (does not reverse when hormones are started/blockers stopped).  The current guidelines recommend the blockers are started before puberty starts and continued for as short a time as possible to give the person certainty of the direction their puberty should progress.  Blockers alone are not meant to be a long-term medication and are not safe for use in this way.  If someone were to use blockers before puberty, remain on T until menopause, and then go off T, there would be no expectation of breast development, however, if they still have ovaries, they may have some minor estrogen-related changes when stopping T completely (because the T levels drop from cis-male range, even if the estrogen levels are also lower than pre-menopause levels).  This would not be too far out of line with the way the body softens with a cis-male in the later ages (and many cis-men develop softening and fat redistribution at andropause, so it wouldn’t be too atypical or give specific cause for being clocked as transgenderAs far as remaining on puberty blockers long term, we don’t have studies that show it’s safe to do this, but we do have indications that suggest there are risks for remaining on them longer than necessary.  you may not be able to find a doctor willing to write for an ongoing (long term) scrip for use in this way.  When you stop, as long as you still have body parts that produce hormones, they would resume making them.  

mod mayhem

[ms-moxxie]

God I cannot wait to be free of Decapeptyl one day. It does its job but it takes a fee

[rayhaber]

Kız Çocuklarında Erken Ergenlik: Zehirli Kimyasalların Etkisi

Kız Çocuklarında Erken Ergenlik: Zehirli Kimyasalların Rolü Dünya genelinde kız çocukları arasında erken ergenlik vakalarının artış göstermesi, sağlık uzmanlarını endişelendiren bir durum haline geldi. Yapılan araştırmalar, bu artışın arkasında yatan nedenlerden birinin zehirli kimyasallara maruz kalma olabileceğini ortaya koymaktadır. Hormonları bozucu özellikleri ile bilinen bazı kimyasallar,…

Kız Çocuklarında Erken Ergenlik: Zehirli Kimyasalların Etkisi

[ricercamix]

Come gli ormoni AMH, GH e IGF1 influenzano l'ipogonadismo ipogonadotropo e la pubertà ritardata

Come gli ormoni AMH, GH e IGF1 influenzano l’ipogonadismo ipogonadotropo e la pubertà ritardata

Mi chiamo Alyssa Paganoni, una studentessa al secondo anno del dottorato in Scienze Farmacologiche Biomolecolari, Sperimentali e Cliniche. Sto svolgendo la mia attività di ricerca presso il laboratorio di Neurobiologia dello Sviluppo, coordinato da Anna Cariboni. Il focus principale del laboratorio è quello di capire i meccanismi cellulari e molecolari che controllano lo sviluppo dei neuroni…

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[pharmaddicted]

Crescere che fatica! I primi segni di uno sviluppo morfologico ed endocrino

Crescere che fatica! I primi segni di uno sviluppo morfologico ed endocrino

Caratteristiche Generali

Il processo di crescita dell’organismo umano deve essere visto come un evento coordinato ed articolato che coinvolge diversi sistemi ed organi. Quando si parla di pubertà ci si riferisce al passaggio dalla fanciullezza allo stato adulto, il tutto è condizionato dall’acquisizione della funzione gonadica e del suo controllo ipotalamo ipofisario. Dunque lo sviluppo…

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[jewishhome-blog]

En una mujer, el sistema de reproducción depende de cinco hormonas principales: progesterona, estrógeno, hormona foliculoestimulante, hormona luteinizante y hormona liberadora de gonadotropina (GnRH). GnRH es la primera hormona liberada por el hipotálamo, una de las regiones del cerebro. Después de ser liberado, la glándula pituitaria produce la hormona estimulante del folículo y la hormona luteinizante. Estas hormonas luego se comunican con los ovarios para permitirles liberar progesterona y estrógeno.