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Sharing some good news. The FDA has approved an oral drug (Xocova) to prevent COVID-19 exposure. One trial showed the drug cut the chance

Sharing some good news.

The FDA has approved an oral drug (Xocova) to prevent COVID-19 exposure. One trial showed the drug cut the chance of getting COVID from a household contact by more than half.

Posted by pubhealthdork on Threads.

#covid #medication #oral drug

What a mass disabling event revealed about medicine’s limits

A great read!

#covid #long covid

The disease has always been tricky to define. A study from Mass General Brigham suggests the healthcare system is missing millions of long C

June 1, 2026 article. Why I strive not to get Covid. I'm the one with the mask in Costco.

#covid #long covid

covid-safer-hotties

Covid caution is workers' rights praxis. Keep your comrades safe whether you work with them or rely upon their labor. Wear a quality respirator in public spaces.

Is Long COVID a real, physical disease with detectable biomarkers? A new study from Yale University and Mount Sinai says: definitely yes, and they have the sick mice to prove it. Researchers found that long COVID patients carry unusual antibodies that attack nervous system tissues, and when those antibodies were injected into healthy mice, the animals developed pain sensitivity, fatigue, and nerve damage strikingly similar to what their human donors reported. This is the clearest evidence yet of a causal mechanism, not merely a correlation, connecting immune system dysfunction to the neurological misery of long COVID.

The findings appear in the journal Cell (doi: 10.1016/j.cell.2026.04.042).

Long COVID affects more than 10% of people who have had a SARS-CoV-2 infection, and women are disproportionately affected. Among the most debilitating complaints are brain fog, chronic pain, dizziness, exhaustion, and memory loss, symptoms that can persist for years and that have largely resisted both clear explanation or effective treatment. The slowness of scientific progress in this area has led to an unjust stigma against people who report symptoms of long COVID.

One leading hypothesis is that COVID triggers the production of autoantibodies: immune proteins that, instead of targeting the virus, mistakenly attack the body’s own cells. This study set out to test whether those rogue antibodies are actually responsible for neurological symptoms, or merely an unrelated symptom.

To investigate, researchers analyzed blood samples from 82 long COVID patients, 30 people who recovered fully from COVID (convalescent controls), and 35 healthy individuals. They found that long COVID patients had elevated levels of autoantibodies that were attacking and damaging a striking range of tissues, including the locus coeruleus and thalamus (brain structures involved in regulating attention, arousal, and sensory processing) as well as peripheral nervous system tissues like the sciatic nerve and the meninges, the membranes surrounding the brain and spinal cord.

The most compelling part of the study was what happened when the scientists extracted purified IgG antibodies from long COVID patients and injected them into healthy mice. Within days, those mice displayed increased sensitivity to heat pain, reduced balance and coordination, muscle weakness, and fatigue, and these behavioral changes tracked closely with the specific symptoms their human donors had reported. When the mice were examined at the cellular level, their peripheral nerve fibers had been damaged, a hallmark of a condition called small fiber neuropathy that is commonly reported in long COVID patients. Brain-wide mapping of neuron activation also lit up regions associated with pain processing, emotional distress, and fatigue. Critically, mice that received antibodies from COVID-recovered individuals without long COVID did not show these effects.

These findings carry important implications for the millions of people still living with long COVID. For a substantial portion of long COVID patients, this is not a psychosomatic condition or a lingering malaise with no physical basis. It is an active autoimmune disease attacking the nervous system, and it deserves treatment as such.

(Drafting of summary accelerated with 🤖 assistance; since the paper is paywalled, it’s important to provide you with a workable digest of the study rather than just the headline)

Posted on Facebook by Catmin's Anticapitalist Treehouse of Solidarity

#covid #long covid

"A nightmare scenario would be if mankind were targeted by a pathogen that attacks our frontal lobes and changes our personalities, making u

"A nightmare scenario would be if mankind were targeted by a pathogen that attacks our frontal lobes and changes our personalities, making us less likely to get along, reach a consensus, and understand others' points of view.....end to society as we know it. Unfortunately, the nightmare may be real and taking shape in SARS-CoV-2, the virus that causes COVID-19."

https://www.infectioncontroltoday.com/view/understanding-impact-covid-19-personality-brain-function-grim-reality-wake-up-call-

Opinion: Kevin Kavanagh, MD, examines a summary of studies on how COVID-19 may damage the brain's frontal lobes, alter personality traits an

#covid #brain function #personality

The immune overlap between long COVID and ME/CFS is not coincidental. It reflects the same underlying dynamic: a viral burden the immune system was never fully able to clear.

Haylie Pomroy and Dr. Rafael Gonzalez discuss why these two conditions share such similar cytokine profiles, immune cell depletion patterns, and clinical presentations, and why the research done on ME/CFS over the past several decades has given clinicians a meaningful head start in understanding

and responding to long COVID.

For far too long, patients with these conditions were dismissed. The science now validates what they have been reporting.

To hear more about the immunological connection between these conditions, tune in to the Hope and Help For Fatigue and Chronic Illness podcast.

URL is below.

The immune overlap between long COVID and ME/CFS is not coincidental. It reflects the same underlying dynamic: a viral burden the immune sys

#CellTherapy #RegenerativeMedicine #LongCOVID

#covid #long covid #mecfs #chronic illness #Youtube

Another great informational post by sam_heds on Instagram.

#covid #sars cov 2 #long covid #t cells #Instagram

A new study from Mass General Brigham found that 1 in 6 people have developed Long Covid. The study used an algorithm to look through medic

A new study from Mass General Brigham found that 1 in 6 people have developed Long Covid.

The study used an algorithm to look through medical records for conditions that were likely related to Long Covid. Some of these conditions included pre-diabetes, heart problems, neurological disorders, fatigue, and chronic pain.

It not only found that Long Covid affected 1 in 6 people, but also that cases of Long Covid increased through mid-2025—and are likely still increasing.

Long COVID may affect roughly 1 in 6 patients based on an AI analysis of electronic health records of nearly 460,000 patients across 58 U.S.

#covid #long covid

Jerry A. Krishnan, MD, PhD, discusses NIH RECOVER findings on long COVID, pulmonary complications, and immune dysfunction at ATS 2026.

Researchers have known what Covid is capable of for years. I think anyone who knows what Covid can do is very tired of trying to warn others.

#covid #long covid #lung damage #immune dysfunction

https://www.threads.com/@80summercamp/post/DYtAfK8Ebxe?xmt=AQG0GyjBJv6cijjd7IG1jK4XRmULFfFHU8Ncck8eYw799zquU-Wt0oxDjxUA6ovYTkkJWtQ&slof=1

Click on the link to see what happens when you get one too many Covid infections.

#covid #long covid

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This young woman explains the dangers of Covid extremely well. I believe her because I too have read so many articles by experts who have tried so hard to warn us

#covid #long covid #Instagram