Fecal Peritonitis As A Result Of Multiple Diverticular Ruptures
The Case
A man of 50 years of age visits our clinic with acute pain in the anal region due to hemorrhoidal disease, at about noon. He mentioned that two (2) weeks before his visit to our clinic, he visited his gastroenterologist who told him to seek surgical consult immediately, but instead he took antibiotics to manage his condition. Upon physical examination, there was a man in pain, with fever, who could not sit due to the pain. Digital examination was impossible since he did not allow it, but the examination revealed an acute inflammation of the hemorrhoid in the 3rd hour, and pus coming out of the anal canal. The patient said he had a mild pain on his left lower quadrant, but nothing special, and he mentioned that the pain was relieved once he defecated.
We admitted the patient, and started hydration, and antibiotic therapy, while painkillers were administered to manage his pain. After almost twelve (12) hours, the clinical picture of the patient changed, with rapid deterioration of his pain. The pain became severe, including in his abdomen, with tenderness, guarding, and rebound tenderness. No bowel movement could be heard, and the fever was present despite the administration of antipyretics and painkillers. An important sign of the deterioration of his clinical condition was the fact that the patient did not wish to proceed to the operation, he now stood quietly on his bed and with a face of agony, asking us to operate on him to “take away his pain”.
Immediately and abdominal CT was ordered, since peritonitis due to ruptured diverticula was our first diagnosis. The CT scan (pic.1 – 3) confirmed our diagnosis, and the patient was prepared for emergency operation.
Pic. 1: edema of the bowel, abscess, and free air in the peritoneal cavity
Pic. 2: CT scanning findings of the patient with peritonitis from diverticular disease.
Pic. 3: free air in the peritoneal cavity, fluid (pus) collection
The findings were impressive, since acute fecal peritonitis was confirmed, with large amounts of pus and feces throughout the peritoneal cavity. After adequate irrigation with many liters of warm natural saline solution of the peritoneal cavity to reduce the microbial and fecal load, symphysiolysis took place, along with the removal of pseudomembranes from the peritoneal reaction.
After mobilization of the sigmoid, the lesions were localized and identified. Multiple ruptures of diverticula in the sigmoid (pic.4). A large amount of the sigmoid was transformed into an abscess (pic. 5), and the disease extended from the sigmoid to descending colon passing the splenic flexure reaching a small portion of the transverse colon. Another impressive finding was the diameter of the ruptured diverticulum at the descending colon (pic. 6). Mobilization of the whole diseased sigmoid and colon was performed, the splenic flexure was mobilized and the colon freed from the spleen and the local symphyses. The part of the transverse colon which was affected was also mobilized and prepared for removal. Indeed, a non oncologic left extended hemicolectomy was performed (pic. 7).
Pic. 4: Rupture lesions from the diverticula
Pic. 5: Sigmoid abscess
Pic. 6: Diameter of the lesion is 3.4cm on the descending colon
Pic. 7: The whole specimen of our non oncologic left extended hemicolectomy, due to diverticular rupture throughout the diseased colon and sigmoid.
After the diseased colon and sigmoid were removed, the operation continued with the anastomosis between the rectum and the transverse colon. An end to end anastomosis was performed with the help of a circular colonic stapler.
Thorough investigation for hemostasis took place. The anastomosis was tested twice for possible leakage. Irrigation of the peritoneal cavity was performed once again.
A loop eileostomy was performed approximately 40 cm from the ileo-cecal valve.
Closure of the abdominal wall was performed with two nylon loop sutures (No.0), and the skin was closed with clips.
The patient recovered from anesthesia in the operating room.
The postoperative course of the patients involved a remarkable recovery from the septic state. On the 2nd postoperative day, the loop ileostomy was functioning, a fact that allowed the patient to start drinking water and tea. Bowel movement was restored on the same day.
The drainage tubes were removed on the 7th day, and on the 8th postoperative day the patient was discharged from the clinic in an excellent condition.
Discussion about this case will follow in the next days.








